213 Matching Annotations
  1. Jan 2020
  2. link.springer.com link.springer.com
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    link.springer.com/article/10.1007/s12031-018-1236-6
  3. www.nature.com www.nature.com
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    nature.com/articles/s41467-019-09477-3
  4. onlinelibrary.wiley.com onlinelibrary.wiley.com
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    onlinelibrary.wiley.com/doi/full/10.1002/glia.23579
  5. Nov 2018
  6. link.springer.com link.springer.com
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    link.springer.com/article/10.1007/s11064-018-2661-1
  7. link.springer.com link.springer.com
    Curcumin Attenuates gp120-Induced Microglial Inflammation by Inhibiting Autophagy via the PI3K Pathway
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    link.springer.com/article/10.1007/s10571-018-0616-3
  8. www.sciencedirect.com www.sciencedirect.com
    Versatility of microglial bioenergetic machinery under starving conditions
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    sciencedirect.com/science/article/pii/S0005272817301937
  9. www.jneurosci.org www.jneurosci.org
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    jneurosci.org/content/37/42/10154
  10. www.sciencedirect.com www.sciencedirect.com
    TREM2 Is a Receptor for β-Amyloid that Mediates Microglial Function
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    sciencedirect.com/science/article/pii/S0896627318300564
  11. europepmc.org europepmc.org
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    europepmc.org/articles/PMC5659819/
  12. Nov 2017
  13. onlinelibrary.wiley.com onlinelibrary.wiley.com
    Inhibition of histone deacetylase 1 or 2 reduces induced cytokine expression in microglia through a protein synthesis independent mechanism
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    onlinelibrary.wiley.com/doi/10.1111/jnc.14144/full
  14. onlinelibrary.wiley.com onlinelibrary.wiley.com
    Ischemia‐responsive protein 94 is a key mediator of ischemic neuronal injury‐induced microglial activation
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    onlinelibrary.wiley.com/doi/10.1111/jnc.14111/full
  15. www.sciencedirect.com www.sciencedirect.com
    TDP-43 Depletion in Microglia Promotes Amyloid Clearance but Also Induces Synapse Loss
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    sciencedirect.com/science/article/pii/S0896627317304750
  16. onlinelibrary.wiley.com onlinelibrary.wiley.com
    The role of autophagy in pro‐inflammatory responses of microglia activation via mitochondrial reactive oxygen species in vitro
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    onlinelibrary.wiley.com/doi/10.1111/jnc.14042/full