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  1. Last 7 days
  2. 54.241.53.176 54.241.53.176
    ssrn-5897399-html.html
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    54.241.53.176/76125d6dcb30a5942befd457cc47a4c8d5cea492/ssrn-5897399-html.html
  3. May 2025
  4. pmc.ncbi.nlm.nih.gov pmc.ncbi.nlm.nih.gov
    Inhibition of SOD1 trimerization is a novel drug target for ALS disease
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    pmc.ncbi.nlm.nih.gov/articles/PMC12067741/
  5. Jan 2025
  6. www.pnas.org www.pnas.org
    Structural insights into the role of reduced cysteine residues in SOD1 amyloid filament formation
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    pnas.org/doi/abs/10.1073/pnas.2408582122
  7. www.nature.com www.nature.com
    Altered mitochondria-associated ER membrane (MAM) function shifts mitochondrial metabolism in amyotrophic lateral sclerosis (ALS)
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    nature.com/articles/s41467-024-51578-1
  8. Apr 2024
  9. www.ncbi.nlm.nih.gov www.ncbi.nlm.nih.gov
    Knockdown of tgfb1a partially improves ALS phenotype in a transient zebrafish model
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    ncbi.nlm.nih.gov/pmc/articles/PMC11032012/
  10. Jul 2023
  11. www.sciencedirect.com www.sciencedirect.com
    Gallic acid inhibits filament formation and promotes the disassembly of superoxide dismutase 1, a protein involved in the pathogenesis of amyotrophic lateral sclerosis
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    sciencedirect.com/science/article/abs/pii/S221242922300593X
  12. Mar 2023
  13. pubs.acs.org pubs.acs.org
    Probing Oxidant Effects on Superoxide Dismutase 1 Oligomeric States in Live Cells Using Single-Molecule Fluorescence Anisotropy
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    pubs.acs.org/doi/full/10.1021/cbmi.3c00002
  14. Oct 2022
  15. www.ncbi.nlm.nih.gov www.ncbi.nlm.nih.gov
    Structural analysis of the overoxidized Cu/Zn-superoxide dismutase in ROS-induced ALS filament formation
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    ncbi.nlm.nih.gov/pmc/articles/PMC9556535/