13 Matching Annotations
  1. Nov 2025
  2. pmc.ncbi.nlm.nih.gov pmc.ncbi.nlm.nih.gov
    The Discovery of Highly Potent THP Derivatives as OCTN2 Inhibitors: From Structure-Based Virtual Screening to In Vivo Biological Activity
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    pmc.ncbi.nlm.nih.gov/articles/PMC7583931/
  3. Jul 2025
  4. pmc.ncbi.nlm.nih.gov pmc.ncbi.nlm.nih.gov
    F2,6BP restores mitochondrial genome integrity in Huntington’s Disease
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    pmc.ncbi.nlm.nih.gov/articles/PMC11580889/
  5. Jan 2025
  6. www.pnas.org www.pnas.org
    Fructose-2,6-bisphosphate restores DNA repair activity of PNKP and ameliorates neurodegenerative symptoms in Huntington's disease
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    pnas.org/doi/10.1073/pnas.2406308121
  7. Jul 2024
  8. www.ncbi.nlm.nih.gov www.ncbi.nlm.nih.gov
    Fructose-2,6-bisphosphate restores DNA repair activity of PNKP and ameliorates neurodegenerative symptoms in Huntington’s disease
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    ncbi.nlm.nih.gov/pmc/articles/PMC10634858/
  9. 52.53.155.43 52.53.155.43
    Table_1-html.html
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    52.53.155.43/178cab1c520cf81e79c8c8f61a8a2e3b091995f9/Table_1-html.html
  10. www.ncbi.nlm.nih.gov www.ncbi.nlm.nih.gov
    HAP40 is a conserved central regulator of Huntingtin and a potential modulator of Huntington’s disease pathogenesis
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    ncbi.nlm.nih.gov/pmc/articles/PMC9295956/
  11. www.ncbi.nlm.nih.gov www.ncbi.nlm.nih.gov
    Assessing the cognitive status of Drosophila by the value-based feeding decision
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    ncbi.nlm.nih.gov/pmc/articles/PMC8443761/
  12. Feb 2024
  13. www.embopress.org www.embopress.org
    A multi‐layered network model identifies Akt1 as a common modulator of neurodegeneration
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    embopress.org/doi/full/10.15252/msb.202311801
  14. Oct 2023
  15. www.mdpi.com www.mdpi.com
    PolyQ-Expansion Causes Mitochondria Fragmentation Independent of Huntingtin and Is Distinct from Traumatic Brain Injury (TBI)/Mechanical Stress-Mediated Fragmentation Which Results from Cell Death
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    mdpi.com/2073-4409/12/19/2406
  16. Jan 2023
  17. www.ncbi.nlm.nih.gov www.ncbi.nlm.nih.gov
    Fullerenols Prevent Neuron Death and Reduce Oxidative Stress in Drosophila Huntington’s Disease Model
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    ncbi.nlm.nih.gov/pmc/articles/PMC9818496/
  18. Sep 2022
  19. www.sciencedirect.com www.sciencedirect.com
    Nucleoporins are degraded via upregulation of ESCRT-III/Vps4 complex in Drosophila models of C9-ALS/FTD
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    sciencedirect.com/science/article/pii/S2211124722012141
  20. Jan 2022
  21. www.sciencedirect.com www.sciencedirect.com
    Transposable element activation promotes neurodegeneration in a Drosophila model of Huntington's disease
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    sciencedirect.com/science/article/pii/S2589004221016722
  22. Apr 2021
  23. www.sciencedirect.com www.sciencedirect.com
    Huntingtin-mediated axonal transport requires arginine methylation by PRMT6
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    sciencedirect.com/science/article/pii/S2211124721002941