11 Matching Annotations
  1. Last 7 days
  2. pmc.ncbi.nlm.nih.gov pmc.ncbi.nlm.nih.gov
    Damage-responsive, maturity-silenced enhancers regulate multiple genes that direct regeneration in Drosophila
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    pmc.ncbi.nlm.nih.gov/articles/PMC7299344/
  3. May 2025
  4. 52.53.155.43 52.53.155.43
    Activation of a Src-JNK pathway in unscheduled endocycling cells of the Drosophila wing disc induces a chronic wounding response
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    52.53.155.43/1a75029db6ee175b9845b4e0ab8095d48576cbfd/NIHPP2025.03.12.642788v1-supplement-1-html.html
  5. Mar 2025
  6. pmc.ncbi.nlm.nih.gov pmc.ncbi.nlm.nih.gov
    Myc and the Tip60 chromatin remodeling complex control neuroblast maintenance and polarity in Drosophila
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    pmc.ncbi.nlm.nih.gov/articles/PMC6092623/
  7. Dec 2024
  8. 52.53.155.43 52.53.155.43
    PMC10780954 adj4457_supplementary_data_1-html.html
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    52.53.155.43/ef2ff34d9f242d15510bd90315d3646309789fb9/PMC10780954-html.html
  9. Aug 2024
  10. 52.53.155.43 52.53.155.43
    PMC6778782 Table S1-html.html
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    52.53.155.43/7224d82ddf0c5a75e2efcc66aacfdf5d4e8da2b0/PMC6778782 Table S1-html.html
  11. Jul 2024
  12. www.ncbi.nlm.nih.gov www.ncbi.nlm.nih.gov
    Wound-induced polyploidization is driven by Myc and supports tissue repair in the presence of DNA damage
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    ncbi.nlm.nih.gov/pmc/articles/PMC6703715/
  13. www.ncbi.nlm.nih.gov www.ncbi.nlm.nih.gov
    Yorkie and JNK revert syncytial muscles into myoblasts during Org-1–dependent lineage reprogramming
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    ncbi.nlm.nih.gov/pmc/articles/PMC6829659/
  14. www.ncbi.nlm.nih.gov www.ncbi.nlm.nih.gov
    Fast drosophila enterocyte regrowth after infection involves a reverse metabolic flux driven by an amino acid transporter
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    ncbi.nlm.nih.gov/pmc/articles/PMC10448536/
  15. 52.53.155.43 52.53.155.43
    PMC10780954 adj4457_supplementary_data_1-html.html
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    52.53.155.43/ef2ff34d9f242d15510bd90315d3646309789fb9/PMC10780954-html.html
  16. Feb 2024
  17. www.nature.com www.nature.com
    Myc plays an important role in Drosophila P-M hybrid dysgenesis to eliminate germline cells with genetic damage
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    nature.com/articles/s42003-020-0923-3