On 2016 Jul 01, Anne Niknejad commented:

'It has been shown that apoptotic stimuli induce nuclear accumulation of GSK3β colocalising it with p53 [15]. This study reported that there was no nuclear accumulation of GSK3β after DNA damage (induced by camptothecin treatment) but rather an exclusive activation of the nuclear pool with no activation of cytosolic pools. The authors showed that p53 coimmunoprecipitates with GSK3β from nuclear fractions after camptothecin treatment.'

Actually the reference 15 is wrong, no 'p53' mention, no camptothecin treatment (but staurosporine treatment)

http://www.ncbi.nlm.nih.gov/pubmed/?term=11495916

The correct reference could be

http://www.ncbi.nlm.nih.gov/pubmed/?term=12048243

(not cited in References)

On 2016 Jul 01, Anne Niknejad commented:

'It has been shown that apoptotic stimuli induce nuclear accumulation of GSK3β colocalising it with p53 [15]. This study reported that there was no nuclear accumulation of GSK3β after DNA damage (induced by camptothecin treatment) but rather an exclusive activation of the nuclear pool with no activation of cytosolic pools. The authors showed that p53 coimmunoprecipitates with GSK3β from nuclear fractions after camptothecin treatment.'

Actually the reference 15 is wrong, no 'p53' mention, no camptothecin treatment (but staurosporine treatment)

http://www.ncbi.nlm.nih.gov/pubmed/?term=11495916

The correct reference could be

http://www.ncbi.nlm.nih.gov/pubmed/?term=12048243

(not cited in References)