- Jul 2018
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europepmc.org europepmc.org
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On 2014 Sep 13, Friedrich Thinnes commented:
A recent study on the phosphorylation status of plasmalemma-integrated type-1 VDAC of neuronal cells under amyloid Aß, from my point of view, brings VDAC-1 back to the field of cell volume regulation and thus apoptosis.
1) Fernandez-Echevarria C, Diaz M, Ferrer I, Canerina-Amaro A, Marin R (2014) Aβ promotes VDAC1 channel dephosphorylation in neuronal lipid rafts. Relevance to the mechanisms of neurotoxicity in Alzheimer's disease. Neuroscience. 2014 Aug 26, in press.
2) Thinnes FP (2014) Opening up of plasmalemma type-1 VDAC to form apoptotic "find me signal" pathways is essential in early apoptosis - evidence from the pathogenesis of cystic fibrosis resulting from failure of apoptotic cell clearance followed by sterile inflammation. Mol Genet Metab. 2014 Apr;111(4):439-44.
This comment, imported by Hypothesis from PubMed Commons, is licensed under CC BY.
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- Feb 2018
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europepmc.org europepmc.org
-
On 2014 Sep 13, Friedrich Thinnes commented:
A recent study on the phosphorylation status of plasmalemma-integrated type-1 VDAC of neuronal cells under amyloid Aß, from my point of view, brings VDAC-1 back to the field of cell volume regulation and thus apoptosis.
1) Fernandez-Echevarria C, Diaz M, Ferrer I, Canerina-Amaro A, Marin R (2014) Aβ promotes VDAC1 channel dephosphorylation in neuronal lipid rafts. Relevance to the mechanisms of neurotoxicity in Alzheimer's disease. Neuroscience. 2014 Aug 26, in press.
2) Thinnes FP (2014) Opening up of plasmalemma type-1 VDAC to form apoptotic "find me signal" pathways is essential in early apoptosis - evidence from the pathogenesis of cystic fibrosis resulting from failure of apoptotic cell clearance followed by sterile inflammation. Mol Genet Metab. 2014 Apr;111(4):439-44.
This comment, imported by Hypothesis from PubMed Commons, is licensed under CC BY.
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