On 2016 Nov 18, David Mage commented:

Any candidate model for SIDS causation must explain the 50% male excess in SIDS and its 4-parameter lognormal age distribution. OMIM shows orexin, pPERK and ATF4 are all autosomal with no X-linkage involved. Therefore this line of research appears to be "barking up the wrong tree." Do the authors have any other explanation for the universal 0.61 male fraction of SIDS other than a recessive X-linkage or pure happenstance?

On 2016 Nov 18, David Mage commented:

Any candidate model for SIDS causation must explain the 50% male excess in SIDS and its 4-parameter lognormal age distribution. OMIM shows orexin, pPERK and ATF4 are all autosomal with no X-linkage involved. Therefore this line of research appears to be "barking up the wrong tree." Do the authors have any other explanation for the universal 0.61 male fraction of SIDS other than a recessive X-linkage or pure happenstance?