332 Matching Annotations
  1. Dec 2019
    1. Ragsdale, D. (2019) Cerebellum, week 10, session 3 notes [PowerPoint slides]. Retrieved from https://mycourses2.mcgill.ca/d2l/le/content/400902/viewContent/4648573/View

      This reference is weak. No one except students registered to this course can view this.

      Presumably this is not the original reference!!

    1. carotid ultrasoundecho-cardiogram

      This is to "determine how the potential stroke is affecting the nervous system"?

      Heart and arteries of the neck? Does not have to do with brain damage?

  2. Nov 2019
    1. up sett

      this was not super clear to me, that it is a group intervention.

      What are the more "direct" benefits? These are a little more "secondary" benefits from your intervention. Start with the direct benefits first!

    1. PPS

      Do people with PPS typically present with mobility problems? Do they look different? Feels that I am missing that information in your booklet. How would I picture someone with PPS? Wheelchair? Walking aid? Normal looking?

    1. motor imagery

      I believe motor imagery exists from various perspectives (imagining yourself doing the movement or looking at yourself). Do they have all the same effect on overt performance?

    1. unload the patient’s bodyweight, facilitating volitional control of gait in patients with neuromuscular diseases like stroke

      expand more on this. Why is this good? Why would it promote VOLITIONAL gait control?

    1. option

      You cover too much ground here. Focus on ONE problem: APAs OR APRs OR dynamic balance OR static balance OR whatever you think works for you and for which you can present depth on the MOTOR LEARNING aspect (ie, NOT strength training).l

    1. By practicing coordination, trunk rotation, and weight shifting during tai chi, this helps elderly gain more control of their own body and react and adjust to unexpected perturbations better

      what kind of exercises are these? why would they be good? why would they reduce fall risk?

    2. deduced that tai chi, compared to any other exercise, results in the most impressive improvements in balance and postural control (Di

      Why could that be? This is getting more relevant.

    1. Figure 9. APRs are involved in the closed-loop (feedback) motor control model (Paquette, 2019)Scalar URLhttp://scalar.usc.edu/works/unilateral-vestibular-loss-and-its-contribution-to-poor-reactive-postural-control/media/feedback-loop (version 4)Source URLhttp://scalar.usc.edu/works/unilateral-vestibular-loss-and-its-contribution-to-poor-reactive-postural-control/media/Feedback%20Loop.png (image/PNG)dcterms:titleFeedback Loopdcterms:descriptionFigure 9. APRs are involved in the closed-loop (feedback) motor control model (Paquette, 2019)View asRDF-XML, RDF-JSON, or HTML

      Schmidt, motor control and learning a behavioral emphasis I believe 2009

    1. n the following video, Lauren, a teenage athlete, explains the symptoms that accompany her vestibular disorder. It is not known whether or not her symptoms are from UVL specifically, but she describes symptoms that would be consistent with UVL.

      Really useful?

    1. 2016

      This section is very good.

      What is your motor impairment exactly? I'm not fully sure? APR in general? APR scaling? APR latency? Postural imbalance? Postural imbalance and APR?

      What is affected in MS with respect to the motor control model?

    2. description

      No reference for your figure. Where did you take it?

      It's very nice but it relates to what part of your text. What does it highlight exactly?

      To me this is an orphan figure.

      Honestly, I would see it at the very beginning of your booklet to explain what ms is. But this is really your call.

    1. Multiple

      this section is very good. Perhaps it would be nice to explain what someone with MS would "look like"?

      You explain what it targets, the immune attacks and types of MS but you do not really tell us what it is.

      Perhaps you could change your figure for something more relevant to what you are describing here? Phases? how demyelination occurs, ms plaques?

      Every time you have a figure you need to relate to it in your text. Figure 1 shows the relationship between...

    1. Some evidence has been shown in support of a genetic cause for PSP. Alterations of the tau gene associated with PSP has been described in various publications (Bonafiti et al. 1999 , Higgins et al. 1999). Additionally, mitochondrial genetic alterations may play a role in the pathogenesis of PSP (Swerdlow et al. 2000, Albers et al. 2001). Unfortunately, no definite answer regarding the cause of PSP has yet to be published. 
      • Careful, this is taken directly from Zampieri and Di Fabio 2006.

      You cannot copy text and just change a word here and there.

      Make sure that there is no "near" copy-paste from the existing literature!!!! Anywhere in your paper.

      I will not look further for similarities in published work. I trust that you will make sure this is not the case in the final version.

    1. ithin 8 hours following the inju

      Drug therapy likely outside the scope of kinesiologists. This is also the acute phase (early in spinal shock) where you would not be doing any treatment for hand recovery.

      Current treatment should be for your motor impairment hand.

    2. A popular and controversial treatment is the use of methylprednisolone (MPSS) (Ahuja et al., 2017, Baptiste & Fehlings, 2007). MPSS is a steroid that aims to reduce oxidative damage caused to neurons by reactive oxygen species that are produced in the healing process following an incomplete SCI (Ahuja et al., 2017). The research done by Ahuja and colleagues (2017) and Baptiste & Fehlings (2007)

      Within those 4 lines you cite the same 2 papers 5 times in total!

    1. Considering the aforementioned muscles innervated by the nerves stemming from the C7-C8 spinal levels,

      Can you synthesize the information you presented previously to make it more digestible and relevant for what you are presenting down the line?

      I do not understand why we have to read about the homonculus for your paper. Does it matter in your case? How important is this?

    1. Diagnosis can be confirmed with computed tomography (CT) and magnetic resonance imaging (MRI) scans (Lee & Thumbikat, 2015). CT scans are used to locate damage in the spinal column by determining fracture stability as illustrated in Figure 4. MRI scans reveal the extent of damage to the spinal cord as seen in Figure 4 a&b.

      What is the difference between a and b?

      What does a show? A ct,really? Is it showing fracture stability as you mention here or is it showing soft tissue damage as well? Why fracture stability and extent of damage? What is the difference and why is this important?

    2. he key dermatomes, as well as muscle strength tests for myotomes

      Feels redundant a bit with what you presented in the first page.

      Again, this is a very dry section that presents very superficial information.

    1. Incomplete SCIs can be contrasted with complete SCIs, which are characterized as a total lack of sensorimotor function beyond the site of injury in the cord.

      Feels like you spend a large chunk of the previous section explaining this. Why do you need to repeat this here?

      Relate to your figure. Why are you presenting this figure?

    1. tic spinal cord injury

      You never really explain what a spinal cord is. You discuss different types and innervation but you don't really say what it is.

      You have key information here but it is not easy to read and it is hard to follow.

      For example, in the first paragraph, except the first sentence you repeat the word incomplete in every sentence. Sometimes, you end your sentence with this word and the first word of the following sentence is again incomplete. You can be much more efficient in you writing and make your text more digestible.

    1. interventions

      You need to work on this section. Ideas are not clearly presented at all, writing is sloppy. What are you trying to do with the wii? Why will it be better than current treatment? How will training progress? What are you expecting from this intervention? Improvements? maintenance?

    2. It is well accepted in the literature that an increase in the quantity of sessions demonstrate greater results (Smits-Engelsman et al, 2012)

      Are you stating the obvious? This was only discovered in 2012??????

    1. habits

      Are you targeting children with this? What age? Is this good for anyone with DCD? Are there different levels of involvement with this pathology? Are some people more dyspraxic than others? Could anyone do the one legged stance on the wii?

      What are you expecting to change in terms of motor control?

    1. On the screen, the console will also show the participants center of pressure; giving them visual feedback on top of the somatosensory and vestibular feedback.

      So it's feedback forever? Is this good?

    2. typically developing (TD) individuals to individuals with DCD using the Yoga tree pose

      Be consistent when you talk about a control group. You use different language throughout your text.

    1. twenty participants with cerebral palsy underwent an 8-week training session using the Table Tilt exercise as a new therapeutic intervention to improve static balance and saw almost immediate improvements. As previously mentioned, DCD is thought to be on the continuum of cerebral palsy; therefore the study may be significantly correlated to how patients with DCD can improve their balance.

      Need to be way more explicit about this.

    1.  Children with DCD are usually diagnosed with other disorders on top of their DCD diagnosis. According to a study done by Harris et al in 2015, 50% of patients diagnosed with DCD will also be diagnosed with ADD or ADHD, as seen on the right, they are all interconnected.DescriptionDetailsCitationsSource fileNo description available.Scalar URLhttp://scalar.usc.edu/works/edkp-3/media/current-therapeutic-techniques (version 1)Source URLhttp://scalar.usc.edu/works/edkp-3/media/dcd.png (image/PNG)dcterms:titleCurrent Therapeutic techniquesView asRDF-XML, RDF-JSON, or HTML  Patients with DCD were also found to have anxiety, language impairments and learning disabilities. Therefore, physical therapy in these cases is not sufficient. Doctors must also properly prescribe appropriate medications and/or psychiatric therapy.

      I already read this in a previous section!!!

    2. Since children with DCD are vision dependant, tasks in eyes closed situations are observed in order to have the child compensate for the loss of vision and practice maintaining their stability.

      You alternate between assessment and treatment. It is confusing. You want to focus on treatment. You spent the motor impairment section talking about what the problem is. Non need to reassess here.

    1. Johnston et al., 2002

      This page is a summary of Jonston et al., 2002. Why not synthesize it with the rest of the postural deficit? Perhaps their finding with the other ones you presented will enable you to make some links?

    1. ed. Th

      This picture comes from a paper that showed that kids are more dependent on visual cues. Are you saying that kids with DCD are even more dependent on visual cues? Would be important to say. You could also tie in with your very first page where you describe multisensory info.

    2. A study done by Wann et al. (1998) observed postural sway in children with DCD. Head movement of the subjects was recorded while standing in the eyes open condition and the closed eyes. It was determined that children with DCD exhibited more postural sway compared to their matched controls when their eyes were closed (Greuze, 2003).

      Good but you can shorten