Degradation ofecosystem services could be significantly slowed down or even reversed if the role ofbiodiversity and its full contribution to economic production were an integrated part ofdecisions made by governmental entities, companies, and other stakeholders (Paul et al2020)20

the reason we say extra virgin olive oil evoo

Fast international travel will, at least temporarily, have to be for urgent or emergency purposes only. A triage approach is needed to ensure that the reallocation of society’s small carbon budget, its labour and resources, are used wisely to provide for a thriving society.

Once decarbonisation is complete, then, if it is considered desirable, rampant inequality can again be pursued, as it clearly is today. But between now and then, inequality is the main obstacle to getting anywhere near our Paris commitments.

Ultra deep geothermal power

fertiliser, the challenge is more real, but there is still an important and obvious first step – eat less meat. A large part of the world’s agricultural system is dedicated to growing crops and vegetables to feed animals, which we then eat. Reduce the last part of this equation (i.e. eat less meat), and the huge inefficiencies in the system mean far less fertiliser is required.

However, CCS on a powerstation is not going to stop CO2 being released from burning kerosene in an aircraft. The only near-medium term answer for this sector is a rapid, massive and fair cut in aviation use – at least until zero-carbon aircraft have replaced most of the current fleet.

When threatened with the possibility of starvation, early humans developed a survival response which sent them foraging for food. Yet foraging is only effective if metabolism is inhibited in various parts of the brain.Foraging requires focus, rapid assessment, impulsivity, exploratory behavior and risk taking. It is enhanced by blocking whatever gets in the way, like recent memories and attention to time. Fructose, a kind of sugar, helps damp down these centers, allowing more focus on food gathering.In fact, the researchers found the entire foraging response was set in motion by the metabolism of fructose whether it was eaten or produced in the body. Metabolizing fructose and its byproduct, intracellular uric acid, was critical to the survival of both humans and animals.The researchers noted that fructose reduces blood flow to the brain’s cerebral cortex involved in self-control, as well as the hippocampus and thalamus. Meanwhile, blood flow increased around the visual cortex associated with food reward. All of this stimulated the foraging response.

Johnson suspects the survival response, what he calls the “survival switch,” that helped ancient humans get through periods of scarcity, is now stuck in the “on” position in a time of relative abundance. This leads to the overeating of high fat, sugary and salty food prompting excess fructose production.Fructose produced in the brain can lead to inflammation and ultimately Alzheimer’s disease, the study said. Animals given fructose show memory lapses, a loss in the ability to navigate a maze and inflammation of the neurons.“A study found that if you keep laboratory rats on fructose long enough they get tau and amyloid beta proteins in the brain, the same proteins seen in Alzheimer’s disease,” Johnson said. “You can find high fructose levels in the brains of people with Alzheimer’s as well.”Johnson suspects that the tendency of some AD patients to wander off might be a vestige of the ancient foraging response.

the numbers are 00:14:09 something like that you drop it down to you've got 88 percent less chance or actually it's it's 12 percent chance for most diseases so most diseases are protected by these diets

eating plants

We’ve been conditioned to view information through aconsumerist lens: that more is better, without limit.

ocio-cultural and lifestyle changes can accelerate climate change mitigation (medium26confidence). Among 60 identified actions that could change individual consumption, individual27mobility choices have the largest potential to reduce carbon footprints. Prioritizing car-free mobility by28walking and cycling and adoption of electric mobility could save 2 tCO2eq cap-1 yr-1. Other options with29high mitigation potential include reducing air travel, cooling setpoint adjustments, reduced appliance30use, shifts to public transit, and shifting consumption towards plant-based diets

diet for typhoid patient

typhoid diet chart

typhoid diet chart in hindi(health and disease)

This idea that plants become desirable or aversive depending on their digestive consequences is simple. But, how do grazing animals figure out exactly which plants made them fee l good or ill? One way herbivores apparently accomplish this task is by regarding unfamiliar plants with caution. Animals associate positive or negative effects of nutrients or toxins with novel foods when offered meals that contain novel and familiar foods. When foraging bouts include several novel plants, plants that dominate the diet are probably 'weighted' more than less-consumed plants, even if the minor foods were primarily responsible for the positive or negative feedback. Furthermore, digestive feedback begins within 10 to 15 minutes of consumption which could help animals attribute digestive benefits or liabilities to specific plants. Finally, livestock grazing on rangelands usually become familiar with the forage resource and may seldom encounter truly novel plants. This allows greater opportunity to 'sort out' feedback from individual or similar groups of plants

When a grazing animal smells and tastes a plant, the flavor is either pleasing or distasteful depending on the animal's previous grazing experiences. When a plant is eaten, it provides feedback during digestion. If consumption of a plant improves the nutrient or energy status of the animal, the plant flavor becomes more desirable or pleasing. If eating of the plant yields illness, the flavor becomes aversive and distasteful (Fig. 2). These flavor-consequence relationships form the basis for dietary likes and dislikes, and the animal then seeks highly palatable foods and avoids aversive foods. The resulting behavior patterns generally lead to increased consump- tion of nutritious foods and limited consumption of toxic or low quality plants.

The Lord led me to a wonderful Christian ophthalmologist with unconventional methods of arresting the disease through diet alone and that has saved my sight.

Following food author Michael Pollan’s simple advice about choosing a diet may be the best way forward: “Eat food. Not too much. Mainly plants.”

One thing that using this tool has highlighted for me is that there are a lot of things happening in our community every day, between news, announcements, events and other stuff. If you only rely on what your social media service of choice has decided is worth knowing because it’s generating clicks or discussion, you’re likely to miss something important. Also, do you really want to get your news crammed in between cat videos and political rants from distant acquaintances?

information diet

1/4 cup (60 mL) ground flax3/4 cup (175 mL) warm water1/2 tsp (2 mL) salt2 tsp (10 mL) canola or flax oil1 3/4 cups (425 mL) all-purpose flour (approx)

The unique Umami characteristic of Dried Shiitake is Guanylate. This Umami component is created during the drying and rehydrating process, and not available in Fresh Shiitake.

Although it has been proposed that NNS do not affect glycemia (3), data from several recent studies suggest that NNS are not physiologically inert. First, it has been demonstrated that the gastrointestinal tract (4,5) and the pancreas (6,7) can detect sugars through taste receptors and transduction mechanisms that are similar to those indentified in taste cells in the mouth. Second, NNS-induced activation of gut sweet taste receptors in isolated duodenal L cells and pancreatic β-cells triggers the secretion of glucagon-like peptide 1 (GLP-1) (4,5) and insulin (6–9), respectively. Third, data from studies conducted in animal models demonstrate that NNS interact with sweet taste receptors expressed in enteroendocrine cells to increase both active and passive intestinal glucose absorption by upregulating the expression of sodium-dependent glucose transporter isoform 1 (5,10,11) and increasing the translocation of GLUT2 to the apical membrane of intestinal epithelia (12).

These variations were related to inflammation in the host

In the first of two enzymatic steps that produce high-glucose syrup as an intermediate to high-fructose syrup and other products, bacterial α-amylase is employed at pH 6–7 and about 105°C to hydrolyze starch purified by wet milling to roughly DE (dextrose equivalent) 10, DE being the percent of reducing end-groups in the aqueous sugar mixture relative to those in pure glucose of the same concentration. This indicates that the average maltooligosaccharide produced has a DP (degree of polymerization) of 10, although the mixture has a very wide DP range. DE 30 or more can be attained if the reaction proceeds to completion, but DE 10 is chosen to minimize the probability of pseudo-crystallization at lower DEs and the extent of alkaline-catalyzed isomerization of the reducing-end glucosyl residue at higher DEs.In the second step, at pH 4.3–4.5 and roughly 60°C, fungal glucoamylase and a small amount of pullulanase (pullulan 6-glucanohydrolase, EC 3.2.1.41), the latter used to rapidly hydrolyze the α-1,6 bonds from the original starch feed, convert the maltooligosaccharide mixture to approximately 96% glucose (dry basis). The remainder is composed of byproducts from α-amylase hydrolysis plus mainly isomaltose [α-glucopyranosyl-(1,6)-glucose] and isomaltotriose from the glucoamylase-catalyzed condensation of glucose.

Extremely easy to follow, the GM Diet Plan for 7 Days offers amazing results. By following this diet plan, one can lose up to 17 pounds of weight within a week.

22.3 per cent (−10.7; 95% CI, −15.6 to −5.7) in the diet group

Sleepiness and neck circumference were significantly reduced only in the diet group (p = .007 and p < .001 for the time × group interactions, respectively).

The sodium-restricted diet group received a regimen aiming a maximum intake of 3 g of sodium per day (equivalent to 7.5 g of sodium chloride).

Alzheimer's disease (AD) is not normally diagnosed until later in life, although evidence suggests that the disease starts at a much earlier age. Risk factors for AD, such as diabetes, hypertension and obesity, are known to have their affects during mid-life, though events very early in life, including maternal over-nutrition, can predispose offspring to develop these conditions. This study tested whether over-nutrition during pregnancy and lactation affected the development of AD in offspring, using a transgenic AD mouse model. Female triple-transgenic AD dam mice (3xTgAD) were exposed to a high-fat (60% energy from fat) or control diet during pregnancy and lactation. After weaning (at 3 weeks of age), female offspring were placed on a control diet and monitored up until 12 months of age during which time behavioural tests were performed. A transient increase in body weight was observed in 4-week-old offspring 3xTgAD mice from dams fed a high-fat diet. However, by 5 weeks of age the body weight of 3xTgAD mice from the maternal high-fat fed group was no different when compared to control-fed mice. A maternal high-fat diet led to a significant impairment in memory in 2- and 12-month-old 3xTgAD offspring mice when compared to offspring from control fed dams. These effects of a maternal high-fat diet on memory were accompanied by a significant increase (50%) in the number of tau positive neurones in the hippocampus. These data demonstrate that a high-fat diet during pregnancy and lactation increases memory impairments in female 3xTgAD mice and suggest that early life events during development might influence the onset and progression of AD later in life.

Objective To determine the effects of diets varying in carbohydrate to fat ratio on total energy expenditure.Design Randomized trial.Setting Multicenter collaboration at US two sites, August 2014 to May 2017.Participants 164 adults aged 18-65 years with a body mass index of 25 or more.Interventions After 12% (within 2%) weight loss on a run-in diet, participants were randomly assigned to one of three test diets according to carbohydrate content (high, 60%, n=54; moderate, 40%, n=53; or low, 20%, n=57) for 20 weeks. Test diets were controlled for protein and were energy adjusted to maintain weight loss within 2 kg. To test for effect modification predicted by the carbohydrate-insulin model, the sample was divided into thirds of pre-weight loss insulin secretion (insulin concentration 30 minutes after oral glucose).Main outcome measures The primary outcome was total energy expenditure, measured with doubly labeled water, by intention-to-treat analysis. Per protocol analysis included participants who maintained target weight loss, potentially providing a more precise effect estimate. Secondary outcomes were resting energy expenditure, measures of physical activity, and levels of the metabolic hormones leptin and ghrelin.Results Total energy expenditure differed by diet in the intention-to-treat analysis (n=162, P=0.002), with a linear trend of 52 kcal/d (95% confidence interval 23 to 82) for every 10% decrease in the contribution of carbohydrate to total energy intake (1 kcal=4.18 kJ=0.00418 MJ). Change in total energy expenditure was 91 kcal/d (95% confidence interval −29 to 210) greater in participants assigned to the moderate carbohydrate diet and 209 kcal/d (91 to 326) greater in those assigned to the low carbohydrate diet compared with the high carbohydrate diet. In the per protocol analysis (n=120, P<0.001), the respective differences were 131 kcal/d (−6 to 267) and 278 kcal/d (144 to 411). Among participants in the highest third of pre-weight loss insulin secretion, the difference between the low and high carbohydrate diet was 308 kcal/d in the intention-to-treat analysis and 478 kcal/d in the per protocol analysis (P<0.004). Ghrelin was significantly lower in participants assigned to the low carbohydrate diet compared with those assigned to the high carbohydrate diet (both analyses). Leptin was also significantly lower in participants assigned to the low carbohydrate diet (per protocol).Conclusions Consistent with the carbohydrate-insulin model, lowering dietary carbohydrate increased energy expenditure during weight loss maintenance. This metabolic effect may improve the success of obesity treatment, especially among those with high insulin secretion

It was supposed to treat severe epilepsy. And as a medical treatment, it was only intended to be administered under the supervision of trained nutritionists and physicians.

Information we receive without consciously asking a question

A beneficial impact of the fat quality on insulin sensitivity is not seen in individuals with a high fat intake (> 37E%).

Though current medical and surgical treatments manage coronary artery disease, they do little to prevent or stop it. Nutritional intervention, as shown in our study and others, has halted and even reversed CAD

The Incredibly Bad Science Behind Dr. Esselstyn’s Plant-based Diet

'The China Study' and 'Forks over Knives', links to critiques

We need to have our crap detectors on high alert, and double-check everything we can, especially if you find it popping back into your mind later.

That means they make it easy to get the “gist” of a “news” story without going to the actual article, and they attract attention away from the name of the source in their news story previews.

Be wary of casual scrolling. This is hard work