328 Matching Annotations
  1. Last 7 days
    1. PMID: 23143947

      PatientID: DIII7

      KindredID: D

      Case: F, 16Y0M, Chinese

      DiseaseAssertion: pancreatic cysts

      FamilyInfo: A 3-generation family is given with 3 confirmed members with this mutation. 2 other members had VHL-presenations however had not been tested for the gene.

      CasePresentingHPOs: HP:0001737 (pancreatic cysts)

      CaseHPOFreeText: initially diagnosed by genetic analysis and further verified radiologically, to be found with pancreatic cysts at 16Y.

      CaseNotHPOs: HP:0010797; HP:0002666; HP:0009711; HP:0005584 (hemangioblastoma, pheochromocytoma, retinal capillary hemangioma, renal cell carcinoma)

      CaseNotHPOFreeText: N/A

      CasePreviousTesting: N/A

      PreviouslyPublished: N/A

      SupplementalData: N/A

      Variant: p.F76del (c.226_228delTTC)

      LegacyVariant: p.F76del (c.439delTCT)

      CaseProblemVariantFreeText: NM_000551.3:c.226_228delTTC automaps to NM_000551.3:c.227_229delTCT

      ClinVarID: N/A

      CAID: CA357012 https://reg.clinicalgenome.org/redmine/projects/registry/genboree_registry/allele?hgvsOrDescriptor=NM_000551.3%3Ac.226_228delTTC

      gnomAD: N/A

      VariantEvidence: N/A

      MutationType: inframe_deletion

      CivicName: F76del (c.227_2289delTCT)

      MultipleGeneVariants: N/A

    1. PatientID: 83 (Cousin)

      KindredID: 18

      Case: F, 50Y0M, Ethnicity Unknown

      DiseaseAssertion: VHL

      FamilyInfo: 11 members of this family tested positive for the c.712C>T/c.499C>T germline variant of the VHL gene. 14 members of this family were assumed to carry this variant due to family history/personal medical history. Members of this family range from asymptomatic to multi-organ involvement (pheochromocytoma, pancreatic and renal cysts/tumors and hemangioblastomas). The family was previously reported in PMID 3942913.

      CasePresentingHPOs: HP:0010797; HP:0009711; HP:0001732 (Hemangioblastoma, Retinal capillary hemangioma, Abnormality of the pancreas)

      CaseHPOFreeText: This patient presented with CNS hemangioblastoma (25Y), retinal angioma (16Y) and pancreatic tumors or cysts (undefined) (22Y).

      CaseNotHPOs: HP:0005584; HP:0000077; HP:0002666 (Renal cell carcinoma, Abnormality of the kidney, Pheochromocytoma)

      CaseNotHPOFreeText: N/A

      CasePreviousTesting: N/A

      PreviouslyPublished: 3942913 (some members of this family are previously reported (phenotypes only) but unable to match patients across papers with confidence)

      SupplementalData: Supplementary Table 1.

      Variant: p.R167W (c.499C>T)

      LegacyVariant: c.712C>T

      CaseProblemVariantFreeText: N/A

      ClinVarID: 2218, https://www.ncbi.nlm.nih.gov/clinvar/variation/2218/

      CAID: N/A

      gnomAD: 0.000007954, https://gnomad.broadinstitute.org/variant/3-10191506-C-T?dataset=gnomad_r2_1

      VariantEvidence: A point mutation (c.712C>T) was found. Being that this variant is out of bound with respect to the NM_000551.3 reference sequence, it was concluded that this variant is noted in the Latif et al. (1993) reference and was translated to c.499C>T (NM_000551.3).

      MutationType: missense_variant;transition

      CivicName: R167W (c.499C>T)

      MultipleGeneVariants: N/A

    2. PatientID: 5 (Aunt)

      KindredID: 17

      Case: F, 38Y0M, Ethnicity Unknown

      DiseaseAssertion: VHL

      FamilyInfo: 6 members of this family tested positive for the c.496_506del germline variant of the VHL gene. ID 4 presented with CNS hemangioblastoma, retinal angioma and renal and pancreatic abnormalities. ID 1, 25 and 8 were asymptomatic.ID 22 presented with CNS hemangioblastoma. 2 members were assumed to have the variant but were not genetically confirmed (ID 20 and ID 11). ID 11 and 20 presented with CNS hemangioblastoma.

      CasePresentingHPOs: HP:0009711 (Retinal capillary hemangioma)

      CaseHPOFreeText: This patient presented with retinal angioma (16Y).

      CaseNotHPOs: HP:0005584; HP:0002666; HP:0010797; HP:0000077; HP:0001732 (Renal cell carcinoma, Pheochromocytoma, Hemangioblastoma, Abnormality of the kidney, Abnormality of the pancreas)

      CaseNotHPOFreeText: N/A

      CasePreviousTesting: N/A

      PreviouslyPublished: N/A

      SupplementalData: Supplementary Table 1.

      Variant: p.V166Sfs*4 (c.496_506del)

      LegacyVariant: N/A

      CaseProblemVariantFreeText: N/A

      ClinVarID: 223229, https://www.ncbi.nlm.nih.gov/clinvar/variation/223229/

      CAID: N/A

      gnomAD: N/A

      VariantEvidence: It is unclear from the publication if this variant is reported according to legacy (AF010238.1) or NM_000551.3 reference sequence. Clinical notes from the case confirm the reference sequence to be NM_000551.3.

      MutationType: deletion; minus_2_frameshift_variant

      CivicName: V166SFS*4 (c.496_506del)

      MultipleGeneVariants: N/A

    1. A female patient presented with VHL disease at age 16, when a symptomatic left adrenal pheochromocytoma was diagnosed and removed laparoscopically. At age 28, a cystic cerebellar hemangioblastoma was removed. Molecular genetic testing revealed the mutation VHL c.500 G > A (p. R167Q).In 2010, she was admitted to the Freiburg Preventive Medicine Center. Baseline MRI detected multiple intraspinal hemangioblastomas associated with tumor cysts. Abdominal MRI also showed a 5.6 × 5.4 cm solid tumor in the pancreatic tail that was richly vascularized

      PatientID: 3

      KindredID: 3

      Case: F, 28Y0M, German

      DiseaseAssertion: VHL

      FamilyInfo: N/A

      CasePresentingHPOs: HP:0006748, HP:0006880, HP:0009713, HP:0030405, HP:0010302 (Adrenal pheochromocytoma, cerebellar hemangioblastoma, spinal hemangioblastoma, pancreatic endocrine tumour, spinal cord tumor)

      CaseHPOFreeText: At age 16 she presented with a left adrenal pheochromocytoma which was removed laparoscopically. At age 28, a cystic cerebellar hemangioblastoma was removed. In 2010, she was admitted to the Freiburg Preventive Medicine Center. Baseline MRI detected multiple intraspinal hemangioblastomas associated with tumor cysts. Abdominal MRI also showed a 5.6 × 5.4 cm solid tumor in the pancreatic tail that was richly vascularized. Three of eleven lymph nodes showed metastases.

      CaseNotHPOs: N/A

      CaseNotHPOFreeText: N/A

      CasePreviousTesting: N/A

      PreviouslyPublished: N/A

      SupplementalData: N/A

      Variant: p.R167Q (c.500G>A)

      LegacyVariant: N/A

      CaseProblemVariantFreeText: N/A

      ClinVar: 2216 https://www.ncbi.nlm.nih.gov/clinvar/variation/2216/

      CAID: N/A

      gnomAD: 0.000003977 https://gnomad.broadinstitute.org/variant/3-10191507-G-A

      VariantEvidence: N/A

      MutationType: missense_variant;transition

      CivicName: R167Q (c.500G>A)

      MultipleGeneVariants: N/A

    1. PMID: 20567917

      PatientID: 2

      KindredID: 2

      Case: F, 29Y0M, Brazilian

      DiseaseAssertion: VHL

      FamilyInfo: VHL type 1. Genetic testing has been performed in her parents and brother with negative results. Her only child has not undergone predictive testing yet.

      CasePresentingHPOs: HP:0030393; HP:0009711; HP:0010797; HP:0000107; HP:0001737; HP:0001407 (endolymphatic sac tumor; retinal capillary hemangioma; hemangioblastoma; renal cysts; pancreatic cysts; hepatic cysts)

      CaseHPOFreeText: endolymphatic sac tumor diagnosed at 11Y, retinal angiomas at 16Y; hemangioblastoma, renal, pancreatic, and hepatic cysts at 29Y

      CaseNotHPOs: N/A

      CaseNotHPOFreeText: N/A

      CasePreviousTesting: N/A

      PreviouslyPublished: N/A

      SupplementalData: Yes

      Variant: p.Q73* (c.217C>T)

      LegacyVariant: N/A

      CaseProblemVariantFreeText: N/A

      ClinVarID: 223164 https://www.ncbi.nlm.nih.gov/clinvar/variation/223164/

      CAID: N/A

      gnomAD: N/A

      VariantEvidence: N/A

      MutationType: stop_gained

      CivicName: Q73* (c.217C>T)

      MultipleGeneVariants: N/A

    1. A deletion of exon 3 in the VHL gene was found in this family.

      PatientID: III-5

      KindredID: Family A

      Case: F, 32Y0M, Ethnicity unknown

      DiseaseAssertion: VHL

      FamilyInfo: Patient’s grandfather (I-1), now deceased, and aunt (II-1) had CNS/spinal hemangioblastoma and the confirmed VHL gene mutation. Her mom II-2 had confirmed VHL and CNS hemangioblastoma; and her cousin (III-6) has renal cell carcinoma and CNS/spinal hemangioblastoma and also the confirmed VHL gene mutation. Her uncle (II-3) has confirmed VHL gene and pancreatic and renal cysts. Her brother and sister (III-3; III-4) both were found with the confirmed VHL gene mutation; III-3 with retinal hemangioblastoma and CNS/spinal hemangioblastomas and III-4 with pancreatic and ovarian cysts. IV-1 and IV-3, were confirmed with the VHL gene mutation although no other manifestations.

      CasePresentingHPOs: HP:0010797; HP:0009711 (CNS hemangioblastoma, retinal capillary hemangioblastoma)

      CaseHPOFreeText: developed hemangioblastoma at the optic nerve in her left eye at the age of 16. At the age of 30, patient A III-5 underwent surgery for a CNS hemangioblastoma.

      CaseNotHPOs: HP:0005584; HP:0002666 (renal cell carcinoma, pheochromocytoma)

      CaseNotHPOFreeText: N/A

      CasePreviousTesting: N/A

      PreviouslyPublished: N/A

      SupplementalData: N/A

      Variant: Deletion of exon 3 (c.464-?_642+?del)

      LegacyVariant: N/A

      CaseProblemVariantFreeText: N/A

      ClinVarID: N/A

      CAID: N/A

      gnomAD: N/A

      VariantEvidence: N/A

      MutationType: exon_loss_variant

      CivicName: Exon 3 Deletion

      MultipleGeneVariants: N/A

    1. We report two young male twins with simultaneously manifesting haemangioblastomas of the right cerebellar hemisphere. In one, the neoplasm was associated with medullary and spinal cord solid tumours; in the other, with a nasopharyngeal bleeding fibroangioma, an association not described previously.

      PatientID: III-2

      KindredID: 1

      Case: M, 16Y0M, ethnicity unknown

      DiseaseAssertion: VHL

      FamilyInfo:  2 young male twins were examined with the VHL gene mutation with simultaneously manifesting hemangioblastomas of the right cerebellar hemisphere. Both parents were examined with no VHL mutation, detecting a de novo mutation in the twins. A third sibling, two years older, presented with a cafè au lait skin spot. Grand mother had bilateral high myopia and glaucoma. The medical history of the pedigree shows that member (I-1) had severe hypertension and died at 60 from a stroke, member (I-2) died at 40 from ovarian carcinoma, and member (I-3) died at 54 from clinically diagnosed intracerebral haemorrhage.

      CasePresentingHPOs: HP:0006880; HP:0009713 (cerebellar hemangioblastoma, spinal hemangioblastoma)

      CaseHPOFreeText: in 1994 patient came complaining of blurred vision thought to be due to his myopia. MRI showed a large cyst and a 4.3 cm mural ovoid nodule in the right cerebellar hemisphere. Another small mural nodule was present in the infero-posterolateral portion of the right cerebellar hemisphere. CT disclosed a round homogeneous, hyperintense nodule at T1 in the spinal cord. Raised intracranial pressure due to the compression of aqueduct from the tumour accounted for hydrocephalus and enlarged lateral and 3rd ventricles.

      CaseNotHPOs: HP:0001901 (polycythemia)

      CaseNotHPOFreeText: N/A

      CasePreviousTesting: N/A

      PreviouslyPublished: N/A

      SupplementalData: N/A

      Variant: p.N78S (c.233A>G)

      CaseProblemVariantFreeText: N/A

      LegacyVariant: p.N78S (c.446A>G)

      ClinVarID: 93326 https://www.ncbi.nlm.nih.gov/clinvar/variation/93326/

      CAID: N/A

      gnomAD: N/A

      VariantEvidence: N/A

      MutationType: missense_variant; transition

      CivicName: N/A

      MultipleGeneVariants: N/A

    1. In 1996, we described a 3 generation VHL 2A kindred with 11 mutation carriers. We aim to share our experience regarding the long-term follow-up of this family

      PatientID: 26

      KindredID: 1

      Case: M, 16Y0M, Ethnicity Unknown

      DiseaseAssertion: VHL

      FamilyInfo: This patient was a member of family 1, where 14/32 total patients were diagnosed with VHL. One of which (I-32) was a mutational carrier with down syndrome. The other 13 patients had varying phenotypes, including adrenal pheochromocytoma, pancreatic endocrine tumors, liver and bone metastases, paraganglioma, cerebellar, spinal, and retinal hemangioblastomas, polycythemia, and chondrosarcoma. See Fig. 1 for family pedigree.

      CasePresentingHPOs: HP:0006748 (adrenal pheochromocytoma)

      CaseHPOFreeText: Patient presented with his initial VHL-related clinical manifestations at 16Y0M. She had adrenal pheochromocytoma and diagnosed with VHL subtype 2A (see table 1).

      CaseNotHPOs: HP:0002668 (paraganglioma)

      CaseNotHPOFreeText: Patient does not have paraganglioma.

      CasePreviousTesting: N/A

      PreviouslyPublished: 8550742

      SupplementalData: N/A

      Variant: p.V166F (c.496G>T)

      LegacyVariant: N/A

      CaseProblemVariantFreeText: N/A

      ClinVarID: 2224, https://www.ncbi.nlm.nih.gov/clinvar/variation/2224/

      CAID: N/A

      gnomAD: N/A

      VariantEvidence: N/A

      MutationType: missense_variant; transversion

      CivicName: V166F (c.496G>T)

      MultipleGeneVariants: N/A

    1. III 10 26 6 PC (15 years) 1 retinal hb (16 years) T c.1-?_340 + ?del

      PatientID: III

      KindredID: 17

      Case: Sex Unknown, 26Y0M, Danish

      DiseaseAssertion: VHL

      FamilyInfo: 2 other family members were found to have this VHL variant. Patient 17-I was asymptomatic. Patient 17-II presented with CNS hemangioblastoma (8Y).

      CasePresentingHPOs: HP:0009711; HP:0001737 (Retinal capillary hemangioma, Pancreatic cysts)

      CaseHPOFreeText: Diagnosed with VHL at age 10Y. Presented with 6 pancreatic cysts (15Y) and retinal hemangioblastoma (16Y). Age at the end of study: 26Y.

      CaseNotHPOs: N/A

      CaseNotHPOFreeText: N/A

      CasePreviousTesting: N/A

      PreviouslyPublished: N/A

      SupplementalData: N/A

      Variant: Exon 1 Deletion (c.1-?_340+?del)

      LegacyVariant: N/A

      CaseProblemVariantFreeText: N/A

      ClinVarID: N/A

      CAID: N/A

      gnomAD: N/A

      VariantEvidence: N/A

      MutationType: exon_loss_variant

      CivicName: Exon 1 Deletion

      MultipleGeneVariants: N/A

    2. 7 I 4 40 3 retinal hb (8 + 16 years) M c. 499 C > T (p.Arg167Trp)

      PatientID: I

      KindredID: 7

      Case: Sex Unknown, 40Y0M, Danish

      DiseaseAssertion: VHL

      FamilyInfo: One other family member presented with this variant and was asymptomatic.

      CasePresentingHPOs: HP:0009711 (Retinal capillary hemangioma)

      CaseHPOFreeText: Diagnosed with VHL at age 4Y. presented with 3 retinal hemangioblastomas (8Y, 16Y). Age at the end of study: 40Y.

      CaseNotHPOs: N/A

      CaseNotHPOFreeText: N/A

      CasePreviousTesting: N/A

      PreviouslyPublished: N/A

      SupplementalData: N/A

      Variant: p.R167W (c.499C>T)

      LegacyVariant: N/A

      CaseProblemVariantFreeText: N/A

      ClinVarID: 2218, https://www.ncbi.nlm.nih.gov/clinvar/variation/2218/

      CAID: N/A

      gnomAD: 0.000007954, https://gnomad.broadinstitute.org/variant/3-10191506-C-T?dataset=gnomad_r2_1

      VariantEvidence: N/A

      MutationType: missense_variant;transition

      CivicName: R167W (c.499C>T)

      MultipleGeneVariants: N/A

    1. A 1 0-year-old boy was admitted to our hospital with complaints of headache and diplopia. On physical examination, he weighed 40 kg (7590p), with a height of 142 cm (50-75p). His brachial blood pressure was 150/120 mmHg (95th percentile 123/81 mmHg). His mother had been operated for bilateral pheochromocytoma at the age of 23 and had not been under medical follow-up since then. Laboratory tests showed elevated 24-hour (h) urinary vanillylmandelic acid (VMA) (40 mg/day; normal value 1-11) and normetanephrine (>2000 µg/day), but normal metanephrine levels (143 µg/day). Abdominal magnetic resonance imaging (MRI) indicated the presence of multiple lesions in perivascular areas neighboring both adrenal glands. An iodine 123 metaiodobenzylguanidine (MIBG) scan revealed multiple focal areas of increased uptake near the adrenal glands. After the preparation for operation, bilateral subtotal adrenalectomy including removal of the masses was performed. The diagnosis of bilateral pheochromocytoma was confirmed histologically and immunohistochemically. The patient remained asymptomatic with no laboratory or radiologic abnormalities for six years of follow-up. At the age of 16, the patient was referred to our clinic with visual defect in the left eye. Ophthalmologic examination led to the diagnosis of bilateral retinal hemangiomas, and he underwent laser photocoagulation. Positive family history with bilateral pheochromocytoma and retinal hemangioma revealed the diagnosis of VHL disease. Cerebral, spinal MRI and abdominal computed tomography (CT) were normal.

      PatientID: Proband

      KindredID: 1

      Case: M, 10Y0M, Ethnicity Unknown

      DiseaseAssertion: VHL

      FamilyInfo: Mother was found with the same VHL germline variant. His mother presented with bilateral pheochromocytoma.

      CasePresentingHPOs: HP:0002315; HP:0000651; HP:0002666; HP:0009711; HP:0000822 (Headache, Diplopia, Pheochromocytoma, Retinal capillary hemangioma, Hypertension)

      CaseHPOFreeText: Admitted with complaints of headache and diplopia. MRI indicated the presence of multiple lesions in perivascular areas neighboring both adrenal glands. An iodine 123 metaiodobenzylguanidine (MIBG) scan revealed multiple focal areas of increased uptake near the adrenal glands. The diagnosis of bilateral pheochromocytoma was confirmed histologically and immunohistochemically. The patient remained asymptomatic with no laboratory or radiologic abnormalities for six years of follow-up. At the age of 16, the patient was referred to our clinic with a visual defect in the left eye. Ophthalmologic examination led to the diagnosis of bilateral retinal hemangiomas, and he underwent laser photocoagulation.

      CaseNotHPOs: HP:0009713; HP:0006880 (Spinal hemangioblastoma, Cerebellar hemangioblastoma)

      CaseNotHPOFreeText: Cerebral, spinal MRI and abdominal computed tomography (CT) were normal.

      CasePreviousTesting: N/A

      PreviouslyPublished: N/A

      SupplementalData: N/A

      Variant: p.R161Q (c.482G>A)

      LegacyVariant: c.695G>A

      CaseProblemVariantFreeText: N/A

      ClinVarID: 182983, https://www.ncbi.nlm.nih.gov/clinvar/variation/182983/

      CAID: N/A

      gnomAD: N/A

      VariantEvidence: Variant was reported as “c.695G>A” which was mapped to p.Arg161Gln (c.482G>A) in accordance with reference sequence NM_000551.3.

      MutationType: missense_variant;transition

      CivicName: R161Q (c.482G>A)

      MultipleGeneVariants: N/A

    1. 16-year-old girl with VHL consisting of retinal and CNS HBs and pancreatic cysts

      PatientID: II-a

      KindredID: 1

      Case: F, 16Y0M, Greek

      DiseaseAssertion: Assumed VHL

      FamilyInfo: The patient’s 50-year-old father has VHL consisting of retinal HBs diagnosed at the age of 20 years. He also has spinal cord HBs located in the neck. He was also found to carry the mutation NM_000551.3:c.433C>T that the patient carried. The patient’s 18-year-old brother and her 20-year-old sister are phenotypically healthy and repeated biochemical evaluation and imaging studies are negative. Her paternal uncle, aunt and cousins are also phenotypically healthy with no indications from either biochemical or imaging studies as they denied further evaluation. In addition, her paternal grandfather of unknown genotype died at the age of 85 years while her paternal grandmother is alive and has no problems, being 86 years old at time of writing.

      CasePresentingHPOs: HP:0009713; HP:0006880; HP:0009711; HP:0001737 (Spinal hemangioblastoma, cerebellar hemangioblastoma, retinal hemangioblastoma, pancreatic cyst)

      CaseHPOFreeText: The 16Y female patient first presented with back pain, headaches, numbness, dizziness, weakness and pain in the arms and the legs at 12Y. A hemangioblastoma in the thoracic spine and a hemangioblastoma in the neck were found. A year later, at the age of 13, multiple HBs in the right and smaller lesions in the left eye were found. Two asymptomatic cysts (2 cm and 0.8 cm) in the body of the pancreas were also found. During her recent evaluation, at the age of 16 years, MRI imaging revealed a large HB extending from the cerebellum to the Th8 vertebrae, with extensive solid and cystic nodules inside the mass and increased dimensions of the spinal cord.

      CaseNotHPOs: N/A

      CaseNotHPOFreeText: N/A

      CasePreviousTesting: N/A

      PreviouslyPublished: N/A

      SupplementalData: N/A

      Variant: p.Q145*(c.433C>T)

      LegacyVariant: N/A

      ClinVarID: 625249, https://www.ncbi.nlm.nih.gov/clinvar/variation/625249/

      gnomAD: N/A

      VariantEvidence: It has been documented that dynamic organisation of pVHL–HIF1α recognition is focally centred on Gln145 within the L7 loop, and its tumourigenic mutant Q145H abolishes almost all of the correlated dynamic motions. In this report, Q145 is substituted by a stop codon, p.Q145X. This is a nonsense mutation resulting to a truncated VHL protein that lacks the last 68 amino acids and thus its function is abolished.

      MutationType: stop_gained

      CivicName: Q145*(c.433C>T)

      MultipleGeneVariants: N/A

    1. F8c.340+617C>G + c.340+648T>C†/WTX1 p.Leu128Val+ X1 p.Leu138Pro/WTNNNNVHL disease

      PatientID: II.6

      KindredID: F8

      Case: F, Age Unknown, Ethnicity Unknown

      DiseaseAssertion: VHL

      FamilyInfo: 6/10 genetically tested family members across two generations were found to harbor the c.340+648T>C and c.340+617C>G germline mutations of the VHL gene. Patient I.2 presented with retinal capillary hemangioma, pheochromocytoma (pheo) and renal cell carcinoma. Patient I.3 presented with CNS hemangioblastoma and pheo. Patient II.1 presented with pheo. Patients II.2, II.6 and II.7 presented with retinal capillary hemangioma.

      CasePresentingHPOs: HP:0009711 (Retinal capillary hemangioma)

      CaseHPOFreeText: Presented with retinal capillary hemangioma. Tumor detected at age 16.

      CaseNotHPOs: N/A

      CaseNotHPOFreeText: N/A

      CasePreviousTesting: N/A

      PreviouslyPublished: N/A

      SupplementalData: N/A

      Variant: c.340+648T>C

      LegacyVariant: N/A

      CaseProblemVariantFreeText: N/A

      ClinVarID: 827583, https://www.ncbi.nlm.nih.gov/clinvar/variation/827583/

      CAID: N/A

      gnomAD: N/A

      VariantEvidence: These variants segregated in 6 patients of this family who developed VHL disease and were absent in 4 healthy tested descendants, indicating their presence on a single disease-associated allele. Sequencing of tumor DNA did not display loss of heterozygosity, suggesting that a WT VHL deletion (as observed in classic VHL disease) may not be a prerequisite in cells of patients with this specific VHL genotype.

      MutationType: missense_variant;transition; splice_site_variant

      CivicName: c.340+617C>G and c.340+648T>C

      MultipleGeneVariants: Germline, VHL, c.340+617C>G

  2. Oct 2026
    1. 4 16.1 Retinal angioma, cervical spine hemangioblastoma, cysts of pancreas and kidney No Germline G to A base change at −1 position of splice acceptor sequence for exon 2

      PatientID: 4

      KindredID: 4

      Case: 16Y1M, sex unknown ethnicity unknown

      DiseaseAssertion: assumed VHL

      FamilyInfo: None

      CasePresentingHPOs: HP:0006880; HP:0009711; HP:0009713; HP:0001737; HP:0000107 (cerebellar hemangioblastoma; retinal capillary hemangioma; spinal hemangioblastoma; pancreatic cysts; renal cyst)

      CaseHPOFreeText: spinal hemangioma was cervical.

      CaseNotHPOs: N/A

      CaseNotHPOFreeText: N/A

      CasePreviousTesting: N/A

      PreviouslyPublished: N/A

      SupplementalData: N/A

      Variant: c.341-1G>A

      CaseProblemVariantFreeText: N/A

      LegacyVariant: N/A

      ClinVarID: 823743 https://www.ncbi.nlm.nih.gov/clinvar/variation/823743/

      CAID: N/A

      gnomAD: N/A

      VariantEvidence: Germline G to A base change at 1 position of splice acceptor sequence for exon 2

      MutationType: splice_site_variant

      CivicName: Splice Site (c.341-1G>A)

      MultipleGeneVariants: N/A

    2. 4 16.1 Retinal angioma, cervical spine hemangioblastoma, cysts of pancreas and kidney No Germline G to A base change at −1 position of splice acceptor sequence for exon 2

      PatientID: 4

      KindredID: 4

      Case: 16Y1M, sex unknown ethnicity unknown

      DiseaseAssertion: assumed VHL

      FamilyInfo: None

      CasePresentingHPOs: HP:0006880; HP:0009711; HP:0009713; HP:0001737; HP:0000107 (cerebellar hemangioblastoma; retinal capillary hemangioma; spinal hemangioblastoma; pancreatic cysts; renal cyst)

      CaseHPOFreeText: spinal hemangioma was cervical.

      CaseNotHPOs: N/A

      CaseNotHPOFreeText: N/A

      CasePreviousTesting: N/A

      PreviouslyPublished: N/A

      SupplementalData: N/A

      Variant: c.341-1G>A

      CaseProblemVariantFreeText: N/A

      LegacyVariant: N/A

      ClinVarID: 823743 https://www.ncbi.nlm.nih.gov/clinvar/variation/823743/

      CAID: N/A

      gnomAD: N/A

      VariantEvidence: Germline G to A base change at 1 position of splice acceptor sequence for exon 2

      MutationType: splice_site_variant

      CivicName: Splice Site (c.341-1G>A)

      MultipleGeneVariants: N/A

    1. GroupID: 42

      DiseaseAssertion: assumed VHL

      GroupSize: 3

      GroupNumberMales: N/A

      GroupNumberFemales: N/A

      GroupEthnicity: N/A

      GroupAgeRange: N/A

      GroupInfo: 3 of the 573 patients discussed in this paper were found to have identical variants. This mutation was found in 1 VHL family with 3 patients.

      GroupPresentingHPOs: HP:0009711; HP:0006880; HP:0002666 (retinal capillary hemangioma, cerebellar hemangioblastoma, pheochromocytoma)

      GroupHPOFreeText: 2 had retinal hemangioblastoma at a mean age of 16Y, one had cerebellar hemangioblastoma at 16Y, and 1 had pheochromcytoma at 14Y.

      GroupNotHPOs: HP:0005584 (renal cell carcinoma)

      GroupNotHPOFreeText: N/A

      GroupPreviousTesting: N/A

      PreviouslyPublished: N/A

      SupplementalData: Supplementary Table S1.

      Variant: c.340+5G>C

      LegacyVariant: N/A

      CaseProblemVariantFreeText: N/A

      ClinVarID: 36901, https://www.ncbi.nlm.nih.gov/clinvar/variation/36901/

      CAID: N/A

      gnomAD: 0.01119 https://gnomad.broadinstitute.org/variant/3-10183876-G-C

      VariantEvidence: This variant’s large allele frequency means this is likely non disease causing (not deleterious).

      MutationType: splice_site_variant

      CivicName: Splice Region (c.340+5G>C)

      MultipleGeneVariants: N/A

    2. GroupID: 42

      DiseaseAssertion: assumed VHL

      GroupSize: 3

      GroupNumberMales: N/A

      GroupNumberFemales: N/A

      GroupEthnicity: N/A

      GroupAgeRange: N/A

      GroupInfo: 3 of the 573 patients discussed in this paper were found to have identical variants. This mutation was found in 1 VHL family with 3 patients.

      GroupPresentingHPOs: HP:0009711; HP:0006880; HP:0002666 (retinal capillary hemangioma, cerebellar hemangioblastoma, pheochromocytoma)

      GroupHPOFreeText: 2 had retinal hemangioblastoma at a mean age of 16Y, one had cerebellar hemangioblastoma at 16Y, and 1 had pheochromcytoma at 14Y.

      GroupNotHPOs: HP:0005584 (renal cell carcinoma)

      GroupNotHPOFreeText: N/A

      GroupPreviousTesting: N/A

      PreviouslyPublished: N/A

      SupplementalData: Supplementary Table S1.

      Variant: c.340+5G>C

      LegacyVariant: N/A

      CaseProblemVariantFreeText: N/A

      ClinVarID: 36901, https://www.ncbi.nlm.nih.gov/clinvar/variation/36901/

      CAID: N/A

      gnomAD: 0.01119 https://gnomad.broadinstitute.org/variant/3-10183876-G-C

      VariantEvidence: This variant’s large allele frequency means this is likely non disease causing (not deleterious).

      MutationType: splice_site_variant

      CivicName: Splice Region (c.340+5G>C)

      MultipleGeneVariants: N/A

  3. Jul 2026
    1. In Kolumbien wird der neugewählte rechte Präsident einen Rollback in der Umweltpolitik und bei der Energiewende einleiten. Er wird dabei auf gut organisierten Widerstand lokaler Communities und der Linken stoßen. Trotz der Politik der Regierung Gustavo Petro ist Kolumbien noch immer von Rohstoffexporten abhängig. Interessierte Machtgruppen setzen vor allem auf die Expansion des Kupfer-Bergbaus.

  4. Apr 2026
  5. Jan 2026
    1. June did this, June did that, she savedmoney and helped clean the house and cookedand Connie couldn't do a thing, her mindwas all filled with trashy daydreams.

      Connie’s mother uses June to compare with Connie, I think her mother wants to take Connie’s pride and her beauty.

  6. Dec 2025
    1. Perhaps his presence gave the evening its peculiar quality of oppressiveness—it stands out in my memory from Gatsby’s other parties that summer.

      Tom’s presence disrupts the carefree atmosphere of Gatsby’s parties and bring tension.

    2. I suppose he smiled at Cody—he had probably discovered that people liked him when he smiled. At any rate Cody asked him a few questions (one of them elicited the brand new name) and found that he was quick and extravagantly ambitious.

      Gatsby learns early on how charm and ambition can help him reshape his identity and move closer to his dreams.

    3. She didn’t like it,” he insisted. “She didn’t have a good time.” He was silent, and I guessed at his unutterable depression. “I feel far away from her,” he said. “It’s hard to make her understand.” “You mean about the dance?” “The dance?” He dismissed all the dances he had given with a snap of his fingers. “Old sport, the dance is unimportant.” He wanted nothing less of Daisy than that she should go to Tom and say: “I never loved you.” After she had obliterated four years with that sentence they could decide upon the more practical measures to be taken. One of them was that, after she was free, they were to go back to Louisville and be married from her house—just as if it were five years ago. “And she doesn’t understand,” he said. “She used to be able to understand. We’d sit for hours—” He broke off and began to walk up and down a desolate path of fruit rinds and discarded favours and crushed flowers.

      yess even gastby sees daisy inside , i don;t get it why tom ignore her

    4. As I went over to say goodbye I saw that the expression of bewilderment had come back into Gatsby’s face, as though a faint doubt had occurred to him as to the quality of his present happiness. Almost five years! There must have been moments even that afternoon when Daisy tumbled short of his dreams—not through her own fault, but because of the colossal vitality of his illusion.

      even nick sees his emotion,he saw the interaction between daisy and gastby

    5. Oh, I’ve been in several things,” he corrected himself. “I was in the drug business and then I was in the oil business. But I’m not in either one now.” He looked at me with more attention. “Do you mean you’ve been thinking over what I proposed the other night?” Before I could answer, Daisy came out of the house and two rows of brass buttons on her dress gleamed in the sunlight. “That huge place there?” she cried pointing. “Do you like it?” “I love it, but I don’t see how you live there all alone.” “I keep it always full of interesting people, night and day. People who do interesting things. Celebrated people.”

      yess they are finally interacting,i can tell gastby pretend to be calm

    6. I suppose he’d had the name ready for a long time, even then. His parents were shiftless and unsuccessful farm people—his imagination had never really accepted them as his parents at all. The truth was that Jay Gatsby of West Egg, Long Island, sprang from his Platonic conception of himself.

      Gatsby rejects his real background and creates a new identity based on an idealized version of himself rather than reality.

    7. I think he was afraid they would dart down a side-street and out of his life forever.

      It is surprising how Tom, who usually dominates every situation, appears genuinely afraid of losing control. His repeated glances back at Gatsby’s car reveal an unexpected vulnerability, as if Daisy might suddenly escape his life altogether.

    8. I think he hardly knew what he was saying

      This sentence reveals how unstable Gatsby’s self-narrative has become. His words no longer sound deliberate but defensive and automatic. It suggests that the persona he performs is slipping beyond his control.

    9. I’d like to just get one of those pink clouds and put you in it and push you around.

      This line shows Daisy’s tendency to turn emotion into decorative fantasy. Her affection is expressed through imagery rather than commitment. It suggests that her love remains playful and unreal, even in an intimate moment.

  7. Nov 2025
    1. June did this, June did that, she savedmoney and helped clean the house and cookedand Connie couldn't do a thing, her mindwas all filled with trashy daydreams.

      I can’t imagine how stress Connie would be if she’d to heard those kind of salty remarks that keep compare her with her sister.

    2. "Stop gawking at yourself. Who are you? You think you're so pretty?" she would say.

      Her mother used the word “gawking”, I think it’s kind of impolite to her daughter. Her mother seemed to be irritated to her that she had to used such an indecent word.

    3. Her mother, who noticed everything and knew everything and whohadn't much reason any longer to look at her own face, always scolded Connie about it.

      It’s interesting here because it’s impossible in real life that one can know everything about the other. But the author made Connie’s mother be a person who omniscient about her daughter.

  8. Oct 2025
    1. He didn't bother talking much to them, but around his bent headConnie's mother kept picking at her until Connie wished her mother was dead and sheherself was dead and it was all over.

      It is surprising that a teenage girl would wish for both her mother and herself to die just because of family arguments.

    2. with charm braceletsjingling on their thin wrists; they would lean together to whisper and laugh secretly ifsomeone passed who amused or interested them. Connie had long dark blond hair thatdrew anyone's eye to it, and she wore part of it pulled up on her head and puffed out andthe rest of it she let fall down her back. She wore a pull-over jersey blouse that looked oneway when she was at home and another way when she was away from home

      This sentenceis interesting because the author writes out the scene which Connie is free and happy with her friends.

  9. Sep 2025
    1. Her heart was almost too bignow for her chest and its pumping made sweat break out all over her. She looked out tosee Arnold Friend pause and then take a step toward the porch, lurching. He almost fell.But, like a clever drunken man, he managed to catch his balance. He wobbled in his highboots and grabbed hold of one of the porch posts.

      This passage is interesting because Arnold Friend seems both clumsy and in control. His strange movements make him look unpredictable and threatening, which makes me feel tense.

    2. Maybe you two better go away," Connie said faintly."What? How come?" Arnold Friend cried. "We come out here to take you for a ride.

      This is troubling because Arnold’s words sound friendly at first, but they actually suggest danger and coercion. It shows that Connie is being lured into a situation she can’t control, creating fear and suspense.

    3. He didn't bother talking much to them, but around his bent headConnie's mother kept picking at her until Connie wished her mother was dead and sheherself was dead and it was all over.

      I'm surprised that Connie and I have the same feeling because of Mom's constant nagging.

    4. mother's tone was approving, and if Connie's name was mentioned it was disapproving.This did not really mean she disliked Connie, and actually Connie thought that hermother preferred her to June just because she was prettier, but the two of them kept up apretense of exasperation, a sense that they were tugging and struggling over something oflittle value to either of them. Sometimes, over coffee, they were almost friends, butsomething would come up—some vexation that was like a fly buzzing suddenly aroundtheir heads—and their faces went hard with contempt.

      Initially, Connie clearly expresses that her mom doesn't like her, but in this paragraph, she says her mom prefers her to June, and sometimes they act like friends. However, they seem to pretend to argue. That is interesting to me.

    5. I took a special interest in you, such a pretty girl, and found out all about you—like I know your parents and sister are gone somewheres and I know where and how longthey're going to be gone, and I know who you were with last night, and your best girlfriend's name is Betty. Right?"

      In the beginning, I think the boy only knows Connie's name, but in this paragraph, he tells her that he knows where her family goes, how long they will be gone, and even the name of her best friend. It makes me feel uncomfortable.

    6. "Connie, you ain't telling the truth. This is your day set aside for a ride with me andyou know it," he said, still laughing. The way he straightened and recovered from his fit oflaughing showed that it had been all fak

      That part where Arnold pretends Connie already agreed to go with him is troubling because it feels manipulative and fake.

    7. n. Ellie turned for the first time and Connie saw withshock that he wasn't a kid either—he had a fair, hairless face, cheeks reddened slightly asif the veins grew too close to the surface of his skin, the face of a forty-year-old ba

      I didn't find that Arnold's friend was even older than him.

    8. He looked at her. He took off the sunglasses and she saw how pale the skin around hiseyes was, like holes that were not in shadow but instead in light. His eyes were like chipsof broken glass that catch the light in an amiable w

      This paragraph vividly portrays Arnold’s image and makes readers sense that something is off about him.

    9. This here is my name, to begin with, he said. ARNOLD FRIENDwas written in tarlike black letters on the side,

      The way he introduced his name was strange, and he even marked numbers on his car, which makes people question his identity and purpose.

    10. They must have been familiar sights, walking around the shopping plaza in theirshorts and flat ballerina slippers that always scuffed the sidewalk,

      This is interesting because it reminds me that teenage girls often pay close attention to their appearance.

    11. "Aunt Tillie's. Right now they're uh—they're drinking. Sitting around," he saidvaguely, squinting as if he were staring all the way to town and over to Aunt Tillie's backyard. Then the vision seemed to get clear and he nodded energetically.

      The stutter gave it away that he's attempting to fabricate a lie. Or that he really had seen them before. It's vaguely put. Either way, it's extremely alarming.

    12. June did this, June did that, she savedmoney and helped clean the house and cookedand Connie couldn't do a thing, her mindwas all filled with trashy daydreams.

      Her mother, to add salt to the wound, would praise her sister, June. Whatever June is Connie isn’t. A distraught favoritism at play.

    13. He looked at her. He took off the sunglasses and she saw how pale the skin around hiseyes was, like holes that were not in shadow but instead in light. His eyes were like chipsof broken glass that catch the light in an amiable way.

      The sharp contrast between his skin suggests he had been chronically predatory.

    14. so much land that Connie had never seen before anddid not recognize except to know that she was going to it.

      An uncertain fate awaits Connie as she is taken away. Whether she really recognize the place or not, the landscape here represents uncertainty, vast and unknown.

    15. Everythingabout her had two sides to it, one for home and one for anywhere that was not home:

      I think this sentence was very interesting is because it shows that Connie is two different people at home and outside. It feels like the common phenomenon among teenagers: "one way in front of parents, another way outside."

  10. drive.google.com drive.google.com
  11. Aug 2025
  12. Jul 2025
  13. Apr 2025
  14. Mar 2025
  15. southtexascollege.blackboard.com southtexascollege.blackboard.com
    1. Though use make you apt to kill me, Let not to that, self-murder added be, And sacrilege, three sins in killing three.

      In line 16 and 17, the speaker essentially tells the listener that if they were to kill the flea, it would be a ot only self-murder, but the murder of three lives: their own, the speaker's, and the flea's. In line 18, the speaker places the flea on this holy pedestal, treating it as if it were sacred . By doing this, he makes it seem as though killing it would be a greater offense than it really is.

  16. Feb 2025
  17. Jan 2025
  18. Nov 2024
  19. Oct 2024
    1. for - rapid whole system change - Nafeez Ahmed - planetary phase shift - Nafeez Ahmed - planetary adaptive cycle - Nafeez Ahmed - essay - The End of Scarcity? From ‘Polycrisis’ to Planetary Phase Shift - Nafeez Ahmed - 2024 Oct 16 - to - book - The Ascent of Humanity - chapter 8 Self and Cosmos: The Gaian Birthing - stillborn and the perilous journey through the womb - Charles Eisenstein

      summary - This is a good article that makes sense of the inflection point that humanity now faces as it contends with multiple existential crisis - It summarizes the complexity of our polycrisis and its precarity and lays the theory for looking at the polycrisis from a different perspective: - as a planetary phase shift towards the potential end of scarcity and the next stage of our species evolution - Through the lens of ecologist Crawford Stanley Holling's lens of the adaptive cycle of ecological population dynamics, - and especially his 2004 paper "From Complex Regions to Complex Worlds" - Nafeez extends Holling's argument that we are undergoing a planetary adaptive cycle in which the back-loop is the dying industrial era. - In this sense, it is reminiscent of the writings of Charles Eisenstein in his book "The Ascent of Humanity", chapter 8: Self and Cosmos:, The Gaian Birth. - Eisenstein uses the the perilous journey of birth through the womb door as a metaphor of the transition we are currently undergoing.

      to - paper - From Complex Regions to Complex Worlds - Crawford Stanley Holling - 2004 - https://hyp.is/KYCm2pFrEe-_PEu84xshXw/www.ecologyandsociety.org/vol9/iss1/art11/main.html?ref=ageoftransformation.org - book - The Ascent of Humanity - Chapter 8 - The Gaian Birthing - Charles Eisenstein - https://hyp.is/r8scTpG_Ee-gLTujlli5hQ/charleseisenstein.org/books/the-ascent-of-humanity/eng/the-gaian-birthing/

    1. Adrian Poisson grew up studying science and math by day and art after hours beginning at the age of five

      for - Adrian Bejan - constructal law - childhood - art and science - from - The End of Scarcity? From ‘Polycrisis’ to Planetary Phase Shift - Nafeez Ahmed - 2024, Oct 16

      Summary - Good explainer video about constructal theory and flow

      from - The End of Scarcity? From ‘Polycrisis’ to Planetary Phase Shift - Nafeez Ahmed - 2024, Oct 16 - https://hyp.is/Qt8IMI74Ee--f4O18QMPFQ/ageoftransformation.org/the-end-of-scarcity-from-polycrisis-to-planetary-phase-shift/

  20. May 2024
    1. Die großen US-Investmentgesellschaften BlackRock, JPMorgan Chase und State Street haben sich zu großen Teilen aus der Koalition Climate Action 100+ zurückgezogen, in der sich Unternehmen zur Dekarbonisierung verpflichten. Damit werden dieser Koalition 14 Billionen (14.000 Millionen) Dollar entzogen. Republikanische Politiker:innen versuchen schon länger zu verhindern, dass Investionen an Dekarbonisierungszusagen gebunden werden. Die Investment-Gesellschaften argumentieren jetzt, dass Climate Action 100+ nicht nur eine Offenlegung fossiler Investionen, sondern eine Steuerung verlangt habe. https://www.nytimes.com/2024/02/16/business/dealbook/wall-streets-climate-retreat.html

    1. Norwegen erteilt in diesem Jahr 62 Lizenzen für die Exploration von Öl- und Gasfeldern, gegenüber 47 im vergangenen Jahr. Die Steigerung geht auf das Interesse von Öl- und Gasgesellschaften zurück. Gegen den Widerstand von NGOs betreibt Norwegen weiterhin eine Ausweitung der Öl- und Gasproduktion, die zu jahrzehntelanger Förderung führen soll. Stark gewachsen ist dabei das Interesse an der Barents Sea. https://www.reuters.com/business/energy/norway-increases-number-new-oil-gas-drilling-permits-including-arctic-2024-01-16/

  21. Apr 2024
  22. Mar 2024
    1. Italien hat in den ersten beiden Monaten dieses Jahres 2,9 Milliarden Kubikmeter Erdgas LMG Transporte erhalten Punkt das sind mehr zum ersten Mal mehr als via Pipeline aus Algerien geliefert wurde. Der der Anteil des russischen erdgases fiel von 40% 20 21 auf 4,7% 2023. Die Infrastruktur für die Anlieferung von LNG wird auf fünf große Anlagen ausgebaut. Insgesamt ist der Gasverbrauch vor allem wegen der milden Winter um 10% gesunken. https://www.repubblica.it/economia/2024/03/16/news/gas_gnl_rigassificatori_gasdotti_russia-422324021/

    1. I have the opposite of low hums, I hear incredibly high-pitched sounds. This video contains one of those frequencies. CRT monitors and other tube TVs are incredibly loud to me even if the volume is off.

      same here, tinnitus at 16 KHz. i guess this comes primarily from the power grid, because when i measure my body skin voltage versus power ground, via microphone port of a smartphone and spectrogram app, then i see the noise around 16 KHz. i guess the source are power electronics connected to the power grid, especially IGBT transistors which have a working frequency around 16 KHz. i spent 2000 euros on EMF shielding for my bedroom (YShield HNV80), but that can only shield high frequencies like 2.4 GHz wifi, but not the low frequencies from the power grid - 50 Hz to 16 KHz. i started hearing this after a LSD trip... expanding ones consciousness in a bad setting is a bad idea

  23. Feb 2024
    1. Preisentwicklungen auf dem Ölmarkt werden zunehmend vom algorithmischen Handel beeinflusst, also von Kauf- und Verkaufsempfehlungen von Computern in Verbindung mit dem Handel von Derivaten. Wie relevant die "Commodity Trading Advisors" (CTAs), die meist computergesteuert und mit regelbasierten Strategen arbeiten, bereits sind, ist umstritten. Hintergrundbericht im Handelsblatt. https://www.handelsblatt.com/finanzen/maerkte/devisen-rohstoffe/ctas-die-unheimliche-macht-auf-dem-oelmarkt-und-was-dahintersteckt/100008536.html

  24. Jan 2024
  25. Dec 2023
    1. Der Standard fasst Berichte der WMO, des europäischen Wetterdienstes Copernicus und des Österreichischen Alpenvereins zum Schwund der Gletscher 2022 zusammen. Die europäischen Alpengletscher verloren 2022 mehr als 5 Kubikkilometer Eis. Die Schweizer Alpengletscher haben in 20 Jahren ein Drittel ihres Eises eingebüßt. Bei einer Temperaturerhöhung um 2,7° bis 2100 würde allein durch das fast vollständige Abschmelzen der Gletscher in Mitteleuropa, den USA, Westkanada und Neuseeland der Meeresspiegel um 115mm steigen.

      https://www.derstandard.de/story/2000145833249/die-gletscher-schmolzen-2022-im-rekordtempo-besonders-in-europa

      Infografik: https://b.staticfiles.at/elm/static/2023-04-schneeanomalie/index.html?nc9238cb923b983a

  26. Nov 2023
    1. And perhaps what students need more than anything for these positive academic habits to flourish is to spend as much time as possible in environments where they feel a sense of belonging, independence, and growth

      Wow, this seems like it would require a drastic change in school environments.

    1. Ein Kommentar in der New York Times weist daraufhin, dass einer der Hauptgründe für das für die geringe Nutzung erneuerbarer Energien die veralteten stromübertragungsnetze sind. In den USA scheitert ihre Erneuerung an den Kosten bzw der mangelnden Bereitschaft, für Sie die Allgemeinheit aufkommen zu lassen Punkt der Artikel erwähnt ähnliche Probleme in Deutschland wo Strom von windkraftwerken im Norden mangelsleitungen nicht nach Süddeutschland transportiert werden kann

  27. Oct 2023
    1. Israel was forbidden to set up sacred stones, pillars: “you shall not set up a pillar (massebah), which the LORD your God hates” (Deuteronomy 16:22).

      Relationship to the first two commandments against worshiping other gods and the use of idols?

      How does this relate to the standing stone found in the room at Khirbet Qeiyafa from the time of David?

      Dates of this text with respect to Khirbet Keiyafa?

    1. Water immobilization is a cool thing! The simplest way to accomplish it is by freezing. But can you think of how water might be immobilized (so to speak) at temperatures above freezing, say at 50°F (10°C)? Think Jell-O and a new process that mimics caviar and you have two methods that nearly stop water in its tracks.

      I learned that science and cooking is always connected. Even if we don't think about it in every day life like when water evaporates or freezes it is chemistry. But what I found most interesting that I learned is how water immobilization works, or to put it more simply the science behind Jell-O. When you add gelatin to water it traps the water molecules in place which creates the sort of liquid and solid hybrid we find with Jell-O.

  28. Sep 2023
  29. Jan 2023
    1. Recommandation 4. Introduire les notions d’éducation à la sexualité dans les programmes officiels decertaines disciplines concernées, au-delà des disciplines liées aux aspects biologiques et sanitaires et del’enseignement moral et civique.Le cadre actuel pose des difficultés liées l’absence de précisions sur l’organisation des séances d’éducation àla sexualité dans l’article L. 312-16 du code de l’éducation, qui a rendu obligatoires au moins trois séancesannuelles en matière d’éducation à la sexualité en 2001. Les circulaires successives ont défini un cadreopérationnel qui a été modifié à plusieurs reprises. Le cadre actuel décrit par la dernière circulaire, en datedu 12 septembre 2018, n’a pas repris deux points sur les supports horaires et les modalités précises sur laprise en charge concrète des séances qui figuraient dans les circulaires de 1998 et 2003. Les modificationssuivantes − sur le modèle de l’article L. 542-3 du code de l’éduction sur l’organisation de la séance annuelled’information et de sensibilisation sur l’enfance maltraitée95 − permettraient de clarifier ces questionsimportantes sur la mise en œuvre concrète de l’EAS :Recommandation 5. Inscrire au moins trois séances annuelles dédiées dans l’emploi du temps des élèves desécoles, des collèges et des lycées (disposition complétant l’article L. 312-16 du code de l’éducation).Recommandation 6. Attribuer la mission d’organisation des séances annuelles aux chefs d’établissement, enlien avec les comités d’éducation à la santé et la citoyenneté (disposition complétant l’article L. 312-16 ducode de l’éducation).

    Tags

    Annotators

  30. Dec 2022
    1. Cette intuition est juste, puisque c’estégalement une approche par la négative queretient la Convention internationale des droitsde l’enfant (CIDE) pour reconnaître, dans sonarticle 16, le droit au respect de la vie privée :« Nul enfant ne fera l’objet d’immixtionsarbitraires ou illégales dans sa vie privée, safamille, son domicile ou sa correspondance,ni d’atteintes illégales à son honneur ou à saréputation »
    2. Cette intuition est juste, puisque c’estégalement une approche par la négative queretient la Convention internationale des droitsde l’enfant (CIDE) pour reconnaître, dans sonarticle 16, le droit au respect de la vie privée :« Nul enfant ne fera l’objet d’immixtionsarbitraires ou illégales dans sa vie privée, safamille, son domicile ou sa correspondance,ni d’atteintes illégales à son honneur ou à saréputation »
    1. Cette intuition est juste, puisque c’estégalement une approche par la négative queretient la Convention internationale des droitsde l’enfant (CIDE) pour reconnaître, dans sonarticle 16, le droit au respect de la vie privée :« Nul enfant ne fera l’objet d’immixtionsarbitraires ou illégales dans sa vie privée, safamille, son domicile ou sa correspondance,ni d’atteintes illégales à son honneur ou à saréputation ».
  31. Nov 2022
    1. The btoa() function takes a JavaScript string as a parameter. In JavaScript strings are represented using the UTF-16 character encoding: in this encoding, strings are represented as a sequence of 16-bit (2 byte) units. Every ASCII character fits into the first byte of one of these units, but many other characters don't. Base64, by design, expects binary data as its input. In terms of JavaScript strings, this means strings in which each character occupies only one byte. So if you pass a string into btoa() containing characters that occupy more than one byte, you will get an error, because this is not considered binary data:
  32. Aug 2022
  33. Mar 2022
  34. Jan 2022
    1. moviepilot.de 5,8/10 IMDB 5,8/10 · 33K · Metascore: 77 Parents Guide

      In den Tiefen des Weltalls, weit entfernt von unserem Sonnensystem, leben Monte (Robert Pattinson) und seine kleine Tochter Willow (Jessie Ross) gemeinsam auf einem ramponierten Raumschiff, dessen Besatzung vor einiger Zeit noch aus vielen verurteilten Schwerverbrechern bestand, die sich mit einer gefährlichen Mission von ihren Strafen freikauften. Mit Experimenten wurden sie von der wahnsinnigen Reproduktionswissenschaftlerin Dibs (Juliette Binoche) gequält, bei denen bis auf Monte und Willow alle ums Leben kamen. Monte ist ein stiller Mann, der sich eine harte Selbstdisziplin auferlegt hat. Doch wenn er mit seiner Tochter zusammen ist, wird aus ihm ein zärtlicher Versorger. Nun sind die beiden die letzten Überlebenden der Crew und nähern sich in völliger Isolation ihrem letzten unausweichlichen Ziel: einem schwarzen Loch und damit auch dem Ende von Zeit und Raum. filmstarts.de

      „High Life“ ist ein schmerzhafter Film, doch es lohnt sich, die Expedition ins Nichts zu begleiten. Wer ein klassisches Weltraum-Epos erleben will, der bleibt besser auf dem Boden. Claire Denis‘ Vision ist kompromisslos und radikal. Ein einzigartiges, schwarzes Juwel. filmstarts.de 4,5/5

      In „High Life“ nimmt uns die französische Arthouse-Regisseurin Claire Denis mit ins Weltall. Von Zivilisation ist da oben aber nichts zu spüren. Stattdessen gibt es eine zwischen Wahnsinn und Klaustrophobie schwankende Stimmung, die nach und nach zwischenmenschliche Abgründe freilegt, während das Publikum vergebens auf Erlösung, Hoffnung oder eine tatsächliche Handlung wartet. Oliver Armknecht 8/10

      An Bord eines Raumschiffes werden übergriffige Experimente durchgeführt, die die Überlebenden vor komplizierte Fragen stellen und dem Publikum schwer zu denken geben. Claire Denis erobert mit ihrem ungewöhnlichen Vertreter eines traditionsreichen Genres reizvolles Neuland. epdFilm ?/10

    1. TMDB (66%) JustWatch (68%) Filmstarts (4/5) Moviepilot (5,4/10) IMDB (5,7/10 · 7,6K | Metascore: 52)

      Handlung

      Ein gemütliches Häuschen auf dem englischen Lande: Der Weihnachtsbaum ist liebevoll geschmückt, das Feiertagsfestmahl opulent vorbereitet und nostalgische Evergreens klingen durch die Räumlichkeit. Als Nell (Keira Knightley), Simon (Matthew Goode) und ihr Sohn Art (Roman Griffin Davis) Verwandtschaft und Freunde in dem kleinen Cottage willkommen heißen, scheint dem perfekten Weihnachtsfest im Kreis der Liebsten rein gar nichts mehr im Wege zu stehen. An der Geschichte gibt es jedoch einen Haken, denn überleben wird niemand die Weihnachtsfeiertage...

      Rezension(en)

      Jüngstes Gericht am Heiligen Abend: Das bitterböse „Was-wäre-wenn“-Kammerspiel von Camille Griffin lässt die Generation „Fridays For Future“ unterm Tannenbaum mit der Dekadenz ihrer Eltern kollidieren – und das ist tatsächlich ein (dunkelschwarzhumoriges) Fest! (filmstarts 4/5)

      „Silent Night“ beginnt wie eine typische Weihnachtskomödie rund um eine dysfunktionale Gruppe, bevor es sich nach und nach in ein Endzeitdrama verwandelt. Die Mischung der einzelnen Bestandteile geht nicht ganz auf, da fehlte dann doch ein schlüssiges Konzept. Sehenswert ist der ungewöhnliche Mix aber, zudem mitreißend gespielt. armknoli

  35. Aug 2021
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  38. Mar 2021
    1. Results for individual PALB2 variants were normalized relative to WT-PALB2 and the p.Tyr551ter (p.Y551X) truncating variant on a 1:5 scale with the fold change in GFP-positive cells for WT set at 5.0 and fold change GFP-positive cells for p.Y551X set at 1.0. The p.L24S (c.71T>C), p.L35P (c.104T>C), p.I944N (c.2831T>A), and p.L1070P (c.3209T>C) variants and all protein-truncating frame-shift and deletion variants tested were deficient in HDR activity, with normalized fold change <2.0 (approximately 40% activity) (Fig. 1a).

      AssayResult: 4.7

      AssayResultAssertion: Normal

      StandardErrorMean: 0.11

    1. Most Suspected Brugada Syndrome Variants Had (Partial) Loss of Function

      AssayResult: 23.2

      AssayResultAssertion: Abnormal

      ReplicateCount: 14

      StandardErrorMean: 7.1

      Comment: This variant had partial loss of function of peak current (10-50% of wildtype) and a >10mV loss of function shift in Vhalf activation, therefore it was considered abnormal (in vitro features consistent with Brugada Syndrome Type 1). (Personal communication: A. Glazer)

  39. Feb 2021
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  43. Jun 2019
    1. chains were generated in the same way as for the isolated a-chains. Thus the model consisted of eight polypeptide chains ( 4 a-chains and 4 13-chains) with the central two a and two 13 chains making up the axial contact interface. The simulations of the above fiber models were carried out without explicit solvent using the GROMOS96 vacuum force field as implemented within the GROMACS suite of programs. Models of HbS mutants were generated using the program SCWRL and the mutant fiber models were generated as for the native HbS model. SCWRL replaces only the side chains of desired residues with the best possible rotamer of the mutated amino acids. Thus the initial backbone conformation of the isolated a-chains and the fiber models remained identical in the native HbS and the mutants. The MD simulation protocol for the fiber models consisted of an initial steepest descent energy minimization for 1000 steps followed by full MD simulation for 1.2ns at 300 K. Essential parameters of the simulation like the radius of gyration, root mean squared deviation from the initial structure as well as the kinetic and potential energies are summarized in Chapter I, Table 4. It was observed that all the global indicators of the simulation stabilized to their average values within 0.2ns. Hence data from 0.2ns till the end of the simulations were used for all subsequent analysis
    2. MD simulations on the a-chain of HbS and the mutants were carried out using the GROMACS suite of programs (Lindahl eta!, 2001). The initial structure of the native protein was taken from the high-resolution x-ray crystal· structure (Harrington et a!, 1997) of HbS (PDB entry: 2HBS). The structures of the mutants were generated interactively using INSIGHT-II. The initial model structures were placed in a simulation box of size 42.8 x 31.5 x 42.7 A. The closest distance from any protein atom to the walls of the box was not less than 9 A. The system was then solvated by adding a bath of SPC (Berendson eta!, 1981) waters in such a way that the density of the system was as close to 1 as possible. The overall charge of the system was neutralized by placing suitable counter ions wherever necessary (Chapter 1, Table 4). The resulting system was then energy minimized for 1000 steps using the steepest descent algorithm. This was followed by 0.3ns of position restrained MD during which the solvent and counter ions were allowed to move freely but the protein atoms were harmonically restrained to their initial positions. Finally, normal MD was run for 3ns using the default GROMACS force field. Bond lengths were restrained to their equilibrium values using the LINCS (Hess et a!, 1997) algorithm and a cut-off radius of 0.9nm was used for non-bonded interaction calculation. The temperature of the system was maintained close to 300K by weak coupling to an external temperature bath with a coupling constant of 0.1 ps. The integration time step used throughout the simulation was 1 fs. MD simulations of single mutant a-chains (K 16Q, E23Q and H20Q) as well as the double mutants (K16Q/H20Q, K16Q/E23Q and H20Q/E23Q) were carried out in a similar fashion for 3ns. In order to directly analyze the effects of the mutations on the contact interface, we also carried out MD simulations on a miniature model of the sickle hemoglobin fiber consisting of a complex of two hemoglobin tetramers. In the low salt crystal structure of deoxyhemoglobin S (Harrington et a!, 1997) the asymmetric unit consists of two HbS tetramers that pack as two strands of HbS molecules running parallel to the crystallographic a axis. Axial contacts occur between two tetramers of the same strand and lateral contacts occur between tetramers of different strands. A canonical fiber model was generated by taking one of the two tetramers in the asymmetric unit together with its neighbor translated along the crystallographic a axis [Figure 7 (A, B)]. Fiber models incorporating mutant a
  44. May 2019
    1. The proteins separated by SDS-PAGE were transferred from the gel to· nitrocellulose membrane using a blotting apparatus (Bio-Rad, USA). In brief, after removal of the stacking gel, the resolving gel was placed over nitrocellulose membrane and sandwiched with Whatman 3 mm filter paper in a cassette. The cassette was submerged in transfer buffer and transfer was carried out at 150 rnA for 3h at 4°C. Following the transfer, the membrane was carefully removed from the blotting apparatus and blocked with 3% non-fat dry milk protein for Ih. The membrane was washed thrice with PBST and incubated overnight with the primary antibody at 4°C. Following incubation, the membrane· was washed thrice with PBST and incubated with appropriate HRP-labeled secondary antibody at room
    1. The phagocytic ability of macrophages was determined by monitoring the uptake of Bioparticles® Alexa fluor 488 labeled dead E. coli (Molecular Probes, Eugene, OR). 2X105 THP-1 macrophages were plated per well in a 24 well plate. Alexa fluor 488 labeled dead E. coli particles were opsonized with an opsonizing reagent obtained from Molecular Probes. These opsonizing reagents are derived from purified rabbit polyclonal IgG antibodies that are specific for E.coli. These opsonized bioparticles® were transferred to the macrophage culture at an multiplicity of infection (MOl) of 1:10, i.e., 10 bacteria per macrophage. The plates were briefly centrifuged at 250 x g to allow the bacteria to settle at the bottom of the plate and were then transferred to an incubator maintained at 37°C and 5% C02 in air for 1 h. The culture medium was aspirated to remove excess unbound bacteria and the cells were washed 3x with ice-cold PBS. To eliminate fluorescence from non-phagocytosed bacteria adhering to the macrophage membrane, 0.25 mg/mL Trypan blue was added and incubated for 10 min to quench the fluorescence of extracellula
    1. Cell-free protein synthesis inhibitory activity of restrictocin was assayed as described by Harlow and Lane ( 1988). The frozen rabbit reticulocyte lysate was thawed on ice in the presence of 30 J..LI of haemin solution per 0.5 ml vial. The toxin was diluted in 0.2% RNase free BSA and several concentrations were incubated with 10 J..LI lysate, I mM ATP, 0.2 mM GTP, 75 mM KCI, 2 mM magnesium acetate, 3 mM glucose, 10 mM Tris-HCI, pH 7.6, 4 J..LM amino acid mixture without leucine, 0.16 J..LCi eH] leucine, 1.33 mg/ml creatine phosphokinase, and 2.66 mg/ml creatine phosphate in a reaction volume of 30 J.LI. The reaction was carried out at 30 °C for one hour and terminated by adding 0.25 ml of I N NaOH containing 0.2% H202• The reaction mixture was further incubated for I 0 min. at 37 °C, BSA added to a final concentration of 65 J..Lg/ml, and the proteins were precipitated with 15% trichloroacetic acid. The mixture was left on ice for 30 min. for complete precipitation and harvested onto 26 mm glass fibre filters. The filter discs were placed in a manifold harvester (millipore) and rinsed with chilled 5% TCA, before the addition of reaction contents. The filters were thoroughly washed with chilled acetone and dried at 37 °C, for one hour. The dried filters were immersed in organic scintillation fluid, and counted using a liquid scintillation counter (Packard).
    1. Following transfection with calcium phosphate or lipofectin, the cells were selected for neomycin resistance by using the analogue G418 Geniticin in the culture medium. After 48 ·hours post -transfection, the cells were harvested and replated at a lower density ( 0.5 x 104 cells I 60 mm dish). Culture medium containing G418 was then added to the cells. G418 was used at two concentrations -400 ug I ml and 500 ug 1 ml. The cells were cultured in G418 containing medium for 2 - 3 weeks. During this period, the mock transfected cells and the cells transfected with plasmid lacking neo gene, died and the transformed cells formed colonies. Individual G418 resistant colonies were picked up and propagated as independent clones. The culture supernates from these clones were analysed by RIA for BhCG. The stability of the BhCG secreting clones was assessed by culturing with several passages over a few weeks in media with or without G418.
    1. RFFIT is used for detennination of rabies virus neutralizing antibody (RVNA) titers. It is an in vitro cell culture based technique in which foci of virus-infected cells are observed by fluorescent antibody staining. In brief, mouse neuroblastoma (MNA) cells were cultured in T-25 tissue culture flasks in DMEM supplemented with 10% FCS at 35°C in humidified atmosphere of0.5% C02. For subculturing, cells were trypsinized (0.5% trypsin + 0.2% EDT A in DMEM without FCS), centrifuged at 150 X g for 10 min, resuspended in DMEM supplemented with 10% FCS and aliquoted into T -25 flasks. Sera from immunized mice were heat inactivated at 56°C for 30 min and the RVNA titers were determined by RFFIT as described previously (Smith . et al., 1996). Briefly, 100 111 of various dilutions of the reference (Standard Rabies Immune Globulin, Biological Research and Reviews, FDA, Maryland, US) and the test sera were mixed with 100 111 Challenge Virus Strain-11 of rabies virus (containing 50 FFD50) in 8-well tissue culture chamber slides and incubated at 35°C in presence of 0.5% C02 for 90 min. After the incubation period, 0.2 ml of MNA cells ( 1 x 1 05) were added to each well and the slides incubated for 40 h following which these were fixed in chilled acetone and stained with FITC conjugated anti-rabies MAb (Centocor Inc, USA) for 45 min. The slides were washed three times with PBS, mounted in glycerol : PBS (9 : 1 ), and examined under fluorescence microscope (Optiphot, Nikon, Japan). Data was expressed as neutralizing antibody titer that is the reciprocal of the serum dilution resulting in a 50% reduction in the number of the virus infected cells in the presence of the test serum.